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Loss of TFB1M results in mitochondrial dysfunction that leads to impaired insulin secretion and diabetes.
Sharoyko, Vladimir V; Abels, Mia; Sun, Jiangming; Nicholas, Lisa M; Mollet, Ines Guerra; Stamenkovic, Jelena A; Göhring, Isabel; Malmgren, Siri; Storm, Petter; Fadista, João; Spégel, Peter; Metodiev, Metodi D; Larsson, Nils-Göran; Eliasson, Lena; Wierup, Nils; Mulder, Hindrik.
Afiliação
  • Sharoyko VV; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Abels M; Unit of Neuroendocrine Cell Biology.
  • Sun J; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Nicholas LM; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Mollet IG; Unit of Islet Cell Exocytosis and.
  • Stamenkovic JA; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Göhring I; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Malmgren S; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Storm P; Unit of Diabetes and Endocrinology, Lund University Diabetes Centre, Clinical Research Centre, Skåne University Hospital, 205 02 Malmö, Sweden and.
  • Fadista J; Unit of Diabetes and Endocrinology, Lund University Diabetes Centre, Clinical Research Centre, Skåne University Hospital, 205 02 Malmö, Sweden and.
  • Spégel P; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism.
  • Metodiev MD; Max Planck Institute for Biology of Ageing, D-50931 Cologne, Germany.
  • Larsson NG; Max Planck Institute for Biology of Ageing, D-50931 Cologne, Germany.
  • Eliasson L; Unit of Islet Cell Exocytosis and.
  • Wierup N; Unit of Neuroendocrine Cell Biology.
  • Mulder H; Department of Clinical Sciences in Malmö, Unit of Molecular Metabolism, hindrik.mulder@med.lu.se.
Hum Mol Genet ; 23(21): 5733-49, 2014 Nov 01.
Article em En | MEDLINE | ID: mdl-24916378
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fatores de Transcrição / Diabetes Mellitus Tipo 2 / Insulina / Mitocôndrias Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans / Male Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2014 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fatores de Transcrição / Diabetes Mellitus Tipo 2 / Insulina / Mitocôndrias Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans / Male Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2014 Tipo de documento: Article