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GOLGA2 loss causes fibrosis with autophagy in the mouse lung and liver.
Park, Sungjin; Kim, Sanghwa; Kim, Min Jung; Hong, Youngeun; Lee, Ah Young; Lee, Hyunji; Tran, Quangdon; Kim, Minhee; Cho, Hyeonjeong; Park, Jisoo; Kim, Kwang Pyo; Park, Jongsun; Cho, Myung-Haing.
Afiliação
  • Park S; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Kim S; Division of Basic Radiation Bioscience, Korea Institute of Radiological & Medical Science, Seoul, Republic of Korea.
  • Kim MJ; Department of Applied Chemistry, College of Applied Science, Kyung Hee University, Yongin 17104, Republic of Korea.
  • Hong Y; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Lee AY; Laboratory of Toxicology, Research Institute for Veterinary Science and College of Veterinary Medicine, Seoul National University, Seoul 08826, Republic of Korea.
  • Lee H; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Tran Q; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Kim M; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Cho H; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Park J; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
  • Kim KP; Department of Applied Chemistry, College of Applied Science, Kyung Hee University, Yongin 17104, Republic of Korea. Electronic address: kimkp@khu.ac.kr.
  • Park J; Department of Pharmacology and Medical Science, Metabolic Syndrome and Cell Signaling Laboratory, Institute for Cancer Research, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea. Electronic address: insulin@cnu.ac.kr.
  • Cho MH; Laboratory of Toxicology, Research Institute for Veterinary Science and College of Veterinary Medicine, Seoul National University, Seoul 08826, Republic of Korea; Graduate School of Convergence Science and Technology, Seoul National University, Suwon 16229, Republic of Korea; Graduate Group of Tumor
Biochem Biophys Res Commun ; 495(1): 594-600, 2018 01 01.
Article em En | MEDLINE | ID: mdl-29128360
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Autoantígenos / Autofagia / Fígado / Cirrose Hepática / Pulmão / Proteínas de Membrana Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Biochem Biophys Res Commun Ano de publicação: 2018 Tipo de documento: Article
Texto completo
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PubMed Links
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Autoantígenos / Autofagia / Fígado / Cirrose Hepática / Pulmão / Proteínas de Membrana Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Biochem Biophys Res Commun Ano de publicação: 2018 Tipo de documento: Article