Microbiota-dependent signals are required to sustain TLR-mediated immune responses.

Weaver, Lehn K; Minichino, Danielle; Biswas, Chhanda; Chu, Niansheng; Lee, Jung-Jin; Bittinger, Kyle; Albeituni, Sabrin; Nichols, Kim E; Behrens, Edward M

Weaver, Lehn K; Minichino, Danielle; Biswas, Chhanda; Chu, Niansheng; Lee, Jung-Jin; Bittinger, Kyle; Albeituni, Sabrin; Nichols, Kim E; Behrens, Edward M.

Afiliação

Weaver LK; Division of Pediatric Rheumatology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.
Minichino D; Cell and Molecular Biology Graduate Group, Perelman School of Medicine, Philadelphia, Pennsylvania, USA.
Biswas C; Division of Pediatric Rheumatology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.
Chu N; Division of Pediatric Rheumatology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.
Lee JJ; CHOP Microbiome Center, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.
Bittinger K; CHOP Microbiome Center, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.
Albeituni S; Department of Oncology, St. Jude Children's Research Hospital, Memphis, Tennessee, USA.
Nichols KE; Department of Oncology, St. Jude Children's Research Hospital, Memphis, Tennessee, USA.
Behrens EM; Division of Pediatric Rheumatology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.

JCI Insight ; 4(1)2019 Jan 10.

Article em En | MEDLINE | ID: mdl-30626747

Palavras-chave

Inflammation; Innate immunity; Monocytes; Mouse models

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: JCI Insight Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Estados Unidos

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