Nicotine induces cell survival and chemoresistance by stimulating Mcl-1 phosphorylation and its interaction with Bak in lung cancer.

Liu, Ling; Shi, Xiaqing; Zhao, Huandong; Yang, Manyi; Wang, Chengzhi; Liao, Mingmei; Zhao, Jinfeng

Liu, Ling; Shi, Xiaqing; Zhao, Huandong; Yang, Manyi; Wang, Chengzhi; Liao, Mingmei; Zhao, Jinfeng.

Afiliação

Liu L; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Shi X; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Zhao H; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Yang M; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Wang C; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Liao M; Department of Nephrology Blood Purification Center, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Zhao J; Key Laboratory of Nanobiological Technology of Chinese Ministry of Health, Xiangya Hospital, Central South University, Changsha, Hunan, China.

J Cell Physiol ; 234(9): 15934-15940, 2019 Sep.

Article em En | MEDLINE | ID: mdl-30741422

Palavras-chave

Mcl-1; chemotherapy sensitivity; lung cancer; nicotine

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: J Cell Physiol Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China

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