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Deficiency of nuclear receptor interaction protein leads to cardiomyopathy by disrupting sarcomere structure and mitochondrial respiration.
Yang, Kai-Chien; Chuang, Kai-Wen; Yen, Won-Shin; Lin, Ssu-Yu; Chen, Hsin-Hsiung; Chang, Szu-Wei; Lin, Yu-Shan; Wu, Wan-Lin; Tsao, Yeou-Ping; Chen, Wen-Pin; Chen, Show-Li.
Afiliação
  • Yang KC; Department and Graduate Institute of Pharmacology, National Taiwan University College of Medicine, Taipei, Taiwan; Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan.
  • Chuang KW; Department and Graduate Institute of Pharmacology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Yen WS; Department and Graduate Institute of Microbiology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Lin SY; Department and Graduate Institute of Microbiology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Chen HH; Department and Graduate Institute of Microbiology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Chang SW; Department and Graduate Institute of Microbiology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Lin YS; Department and Graduate Institute of Pharmacology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Wu WL; Department and Graduate Institute of Pharmacology, National Taiwan University College of Medicine, Taipei, Taiwan.
  • Tsao YP; Department of Ophthalmology, Mackay Memorial Hospital, Taipei, Taiwan.
  • Chen WP; Department and Graduate Institute of Pharmacology, National Taiwan University College of Medicine, Taipei, Taiwan. Electronic address: chenwenpin@ntu.edu.tw.
  • Chen SL; Department and Graduate Institute of Microbiology, National Taiwan University College of Medicine, Taipei, Taiwan. Electronic address: showlic@ntu.edu.tw.
J Mol Cell Cardiol ; 137: 9-24, 2019 12.
Article em En | MEDLINE | ID: mdl-31629737
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Sarcômeros / Proteína 1 de Interação com Receptor Nuclear / Mitocôndrias Cardíacas / Cardiomiopatias Tipo de estudo: Prognostic_studies Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Taiwan
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Sarcômeros / Proteína 1 de Interação com Receptor Nuclear / Mitocôndrias Cardíacas / Cardiomiopatias Tipo de estudo: Prognostic_studies Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2019 Tipo de documento: Article País de afiliação: Taiwan