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CaMKII exacerbates heart failure progression by activating class I HDACs.
Zhang, Manling; Yang, Xue; Zimmerman, Raymond J; Wang, Qin; Ross, Mark A; Granger, Jonathan M; Luczak, Elizabeth D; Bedja, Djahida; Jiang, Hong; Feng, Ning.
Afiliação
  • Zhang M; Department of Medicine, Division of Cardiology, Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA, United States; Division of Cardiology, Veteran Affair Pittsburgh Healthcare System, Pittsburgh, PA, United States.
  • Yang X; Department of Medicine, Division of Cardiology, Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA, United States; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Zimmerman RJ; Department of Medicine, Division of Cardiology, Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA, United States.
  • Wang Q; Department of Medicine, Division of Cardiology, Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA, United States; Echocardiography lab at Heart Center, Ningxia General Hospital, Ningxia Medical University, Ningxia, China.
  • Ross MA; Center for Biologic Imaging, University of Pittsburgh, Pittsburgh, PA, United States of America.
  • Granger JM; Division of Cardiology, Department of Medicine, Johns Hopkins University, Baltimore, MD, United States.
  • Luczak ED; Division of Cardiology, Department of Medicine, Johns Hopkins University, Baltimore, MD, United States.
  • Bedja D; Division of Cardiology, Department of Medicine, Johns Hopkins University, Baltimore, MD, United States.
  • Jiang H; Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, China.
  • Feng N; Department of Medicine, Division of Cardiology, Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA, United States; Division of Cardiology, Veteran Affair Pittsburgh Healthcare System, Pittsburgh, PA, United States. Electronic address: fengn@pitt.edu.
J Mol Cell Cardiol ; 149: 73-81, 2020 12.
Article em En | MEDLINE | ID: mdl-32971072
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Progressão da Doença / Proteína Quinase Tipo 2 Dependente de Cálcio-Calmodulina / Insuficiência Cardíaca / Histona Desacetilases Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2020 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Progressão da Doença / Proteína Quinase Tipo 2 Dependente de Cálcio-Calmodulina / Insuficiência Cardíaca / Histona Desacetilases Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2020 Tipo de documento: Article País de afiliação: Estados Unidos