CstF64-Induced Shortening of the <i>BID</i> 3'UTR Promotes Esophageal Squamous Cell Carcinoma Progression by Disrupting ceRNA Cross-talk with <i>ZFP36L2</i>.

Lin, Ai; Ji, Ping; Niu, Xiangjie; Zhao, Xuan; Chen, Yamei; Liu, Weiling; Liu, Yachen; Fan, Wenyi; Sun, Yanxia; Miao, Chuanwang; Zhang, Shaosen; Tan, Wen; Lin, Dongxin; Wagner, Eric J; Wu, Chen

CstF64-Induced Shortening of the BID 3'UTR Promotes Esophageal Squamous Cell Carcinoma Progression by Disrupting ceRNA Cross-talk with ZFP36L2.

Lin, Ai; Ji, Ping; Niu, Xiangjie; Zhao, Xuan; Chen, Yamei; Liu, Weiling; Liu, Yachen; Fan, Wenyi; Sun, Yanxia; Miao, Chuanwang; Zhang, Shaosen; Tan, Wen; Lin, Dongxin; Wagner, Eric J; Wu, Chen.

Afiliação

Lin A; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Ji P; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, Texas.
Niu X; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, Texas.
Zhao X; Fulgent Genetics, Houston, Texas.
Chen Y; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Liu W; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Liu Y; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Fan W; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Sun Y; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Miao C; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Zhang S; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Tan W; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Lin D; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Wagner EJ; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Wu C; Department of Etiology and Carcinogenesis, National Cancer Center/National Clinical Research Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.

Cancer Res ; 81(22): 5638-5651, 2021 11 15.

Article em En | MEDLINE | ID: mdl-34607841

Assuntos

Regiões 3' não Traduzidas/genética; Proteína Agonista de Morte Celular de Domínio Interatuante com BH3/genética; Fator Estimulador de Clivagem/metabolismo; Neoplasias Esofágicas/patologia; Carcinoma de Células Escamosas do Esôfago/patologia; Regulação Neoplásica da Expressão Gênica; Fatores de Transcrição/metabolismo; Animais; Apoptose; Biomarcadores Tumorais/genética; Biomarcadores Tumorais/metabolismo; Proliferação de Células; Fator Estimulador de Clivagem/genética; Neoplasias Esofágicas/genética; Neoplasias Esofágicas/metabolismo; Carcinoma de Células Escamosas do Esôfago/genética; Carcinoma de Células Escamosas do Esôfago/metabolismo; Humanos; Masculino; Camundongos; Camundongos Endogâmicos NOD; Camundongos SCID; Poliadenilação; Prognóstico; RNA-Seq; Taxa de Sobrevida; Fatores de Transcrição/genética; Transcriptoma; Células Tumorais Cultivadas; Sequenciamento do Exoma; Ensaios Antitumorais Modelo de Xenoenxerto

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fatores de Transcrição / Neoplasias Esofágicas / Regulação Neoplásica da Expressão Gênica / Regiões 3' não Traduzidas / Fator Estimulador de Clivagem / Proteína Agonista de Morte Celular de Domínio Interatuante com BH3 / Carcinoma de Células Escamosas do Esôfago Tipo de estudo: Prognostic_studies Limite: Animals / Humans / Male Idioma: En Revista: Cancer Res Ano de publicação: 2021 Tipo de documento: Article País de afiliação: China

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