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Interplay between IL-10, IFN-γ, IL-17A and PD-1 Expressing EBNA1-Specific CD4+ and CD8+ T Cell Responses in the Etiologic Pathway to Endemic Burkitt Lymphoma.
Forconi, Catherine S; Mulama, David H; Saikumar Lakshmi, Priya; Foley, Joslyn; Otieno, Juliana A; Kurtis, Jonathan D; Berg, Leslie J; Ong'echa, John M; Münz, Christian; Moormann, Ann M.
Afiliação
  • Forconi CS; Division of Infectious Diseases and Immunology, Department of Medicine, University of Massachusetts Chan Medical School, Worcester, MA 01605, USA.
  • Mulama DH; Center for Global Health Research, Kenya Medical Research Institute, Kisumu 40100, Kenya.
  • Saikumar Lakshmi P; Division of Infectious Diseases and Immunology, Department of Medicine, University of Massachusetts Chan Medical School, Worcester, MA 01605, USA.
  • Foley J; Division of Infectious Diseases and Immunology, Department of Medicine, University of Massachusetts Chan Medical School, Worcester, MA 01605, USA.
  • Otieno JA; Jaramogi Oginga Odinga Teaching and Referral Hospital, Ministry of Medical Services, Kisumu 40100, Kenya.
  • Kurtis JD; Center for International Health Research, Department of Pathology and Laboratory Medicine, Rhode Island Hospital, The Warren Alpert Medical School of Brown University, Providence, RI 02903, USA.
  • Berg LJ; Department of Immunology and Microbiology, University of Colorado, Anschutz Medical Campus, Aurora, CO 80045, USA.
  • Ong'echa JM; Center for Global Health Research, Kenya Medical Research Institute, Kisumu 40100, Kenya.
  • Münz C; Department of Viral Immunobiology, Institute of Experimental Immunology, University of Zürich, CH-8057 Zurich, Switzerland.
  • Moormann AM; Division of Infectious Diseases and Immunology, Department of Medicine, University of Massachusetts Chan Medical School, Worcester, MA 01605, USA.
Cancers (Basel) ; 13(21)2021 Oct 27.
Article em En | MEDLINE | ID: mdl-34771539
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Etiology_studies Idioma: En Revista: Cancers (Basel) Ano de publicação: 2021 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Etiology_studies Idioma: En Revista: Cancers (Basel) Ano de publicação: 2021 Tipo de documento: Article País de afiliação: Estados Unidos