TLR4 Associated Signaling Disrupters as a New Means to Overcome HERV-W Envelope-Mediated Myelination Deficits.

Göttle, Peter; Schichel, Kira; Reiche, Laura; Werner, Luisa; Zink, Annika; Prigione, Alessandro; Küry, Patrick

Göttle, Peter; Schichel, Kira; Reiche, Laura; Werner, Luisa; Zink, Annika; Prigione, Alessandro; Küry, Patrick.

Afiliação

Göttle P; Department of Neurology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Schichel K; Department of Neurology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Reiche L; Department of Neurology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Werner L; Department of Neurology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Zink A; Department of General Pediatrics, Neonatology and Pediatric Cardiology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Prigione A; Department of General Pediatrics, Neonatology and Pediatric Cardiology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.
Küry P; Department of Neurology, Medical Faculty, Heinrich-Heine-University, Düsseldorf, Germany.

Front Cell Neurosci ; 15: 777542, 2021.

Article em En | MEDLINE | ID: mdl-34887730

Palavras-chave

ENV; HERV-W; endogenous retrovirus; mitochondrial dysfunction; multiple sclerosis (MS); myelin repair; oligodendrocyte; toll-like receptor

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Risk_factors_studies Idioma: En Revista: Front Cell Neurosci Ano de publicação: 2021 Tipo de documento: Article País de afiliação: Alemanha

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