miR-196a enhances polymerization of neuronal microfilaments through suppressing IMP3 and upregulating IGF2 in Huntington's disease.

Yang, Han-In; Huang, Pin-Yu; Chan, Siew Chin; Tung, Chih-Wei; Cheng, Pei-Hsun; Chen, Chuan-Mu; Yang, Shang-Hsun

Yang, Han-In; Huang, Pin-Yu; Chan, Siew Chin; Tung, Chih-Wei; Cheng, Pei-Hsun; Chen, Chuan-Mu; Yang, Shang-Hsun.

Afiliação

Yang HI; Department of Physiology, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Huang PY; Department of Physiology, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Chan SC; Department of Physiology, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Tung CW; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Cheng PH; Department of Physiology, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Chen CM; Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.
Yang SH; Department of Physiology, College of Medicine, National Cheng Kung University, Tainan 70101, Taiwan.

Mol Ther Nucleic Acids ; 30: 286-299, 2022 Dec 13.

Article em En | MEDLINE | ID: mdl-36320323

Palavras-chave

Huntington's disease; IGF2; IMP3; MT: Non-coding RNAs; miR-196a; microfilament; neurite outgrowth

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Mol Ther Nucleic Acids Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Taiwan

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