Extracellular matrix and cyclic stretch alter fetal cardiomyocyte proliferation and maturation in a rodent model of heart hypoplasia.

Watson, Matthew C; Williams, Corin; Wang, Raymond M; Perreault, Luke R; Sullivan, Kelly E; Stoppel, Whitney L; Black, Lauren D

Watson, Matthew C; Williams, Corin; Wang, Raymond M; Perreault, Luke R; Sullivan, Kelly E; Stoppel, Whitney L; Black, Lauren D.

Afiliação

Watson MC; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.
Williams C; Department of Mechanical Engineering, Tufts University, Medford, MA, United States.
Wang RM; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.
Perreault LR; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.
Sullivan KE; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.
Stoppel WL; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.
Black LD; Department of Biomedical Engineering, Tufts University, Medford, MA, United States.

Front Cardiovasc Med ; 9: 993310, 2022.

Article em En | MEDLINE | ID: mdl-36518682

Palavras-chave

cardiomyocytes; extracellular matrix (ECM); heart hypoplasia; mechanical stretch; proliferation

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Idioma: En Revista: Front Cardiovasc Med Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Estados Unidos

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