Src-FAK Signaling Mediates Interleukin 6-Induced HCT116 Colorectal Cancer Epithelial-Mesenchymal Transition.

Huang, Yu-Han; Chen, Han-Kun; Hsu, Ya-Fen; Chen, Hsiu-Chen; Chuang, Chin-Hui; Huang, Shiu-Wen; Hsu, Ming-Jen

Huang, Yu-Han; Chen, Han-Kun; Hsu, Ya-Fen; Chen, Hsiu-Chen; Chuang, Chin-Hui; Huang, Shiu-Wen; Hsu, Ming-Jen.

Afiliação

Huang YH; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Chen HK; Department of General Surgery, Chi Mei Medical Center, Tainan 710, Taiwan.
Hsu YF; Division of General Surgery, Department of Surgery, Landseed Hospital, Taoyuan 324, Taiwan.
Chen HC; Department of Pharmacology, School of Medicine, College of Medicine, Taipei Medical University, Taipei 110, Taiwan.
Chuang CH; Graduate Institute of Medical Sciences, College of Medicine, Taipei Medical University, Taipei 110, Taiwan.
Huang SW; Department of Pharmacology, School of Medicine, College of Medicine, Taipei Medical University, Taipei 110, Taiwan.
Hsu MJ; Graduate Institute of Medical Sciences, College of Medicine, Taipei Medical University, Taipei 110, Taiwan.

Int J Mol Sci ; 24(7)2023 Apr 02.

Article em En | MEDLINE | ID: mdl-37047623

Assuntos

Neoplasias Colorretais; Interleucina-6; Humanos; Linhagem Celular Tumoral; Movimento Celular; Transição Epitelial-Mesenquimal/genética; Interleucina-6/metabolismo; Transdução de Sinais; Genes src

Palavras-chave

Src; colorectal cancer; epithelialmesenchymal transition (EMT); focal adhesion kinase (FAK); interleukin-6 (IL-6); signal transducer and activator of transcription 3 (STAT3)

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Neoplasias Colorretais / Interleucina-6 Limite: Humans Idioma: En Revista: Int J Mol Sci Ano de publicação: 2023 Tipo de documento: Article País de afiliação: Estados Unidos

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