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1.
Tafazzin deficiency in mouse mesenchymal stem cells promote reprogramming of activated B lymphocytes toward immunosuppressive phenotypes.
FASEB J
; 36(8): e22443, 2022 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-35816277
2.
Critical Roles of Phosphoinositide 3-Kinase δ in the Humoral Immune Response to Trypanosoma congolense Infection.
J Immunol
; 207(5): 1401-1410, 2021 09 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-34380646
3.
Dual inhibition of phosphoinositide 3-kinases delta and gamma reduces chronic B cell activation and autoantibody production in a mouse model of lupus.
Front Immunol
; 14: 1115244, 2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-37234154
4.
Crosspteryx fibrifuga leaf extract enhances host resistance to Trypanosoma congolense infection in mice by regulating host immune response and disrupting the activity of parasite superoxide dismutase enzyme.
Front Microbiol
; 14: 1275365, 2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-37954253
5.
Tafazzin deficiency in mouse mesenchymal stem cells potentiates their immunosuppression and impairs activated B lymphocyte immune function.
bioRxiv
; 2021 Sep 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-34729562
6.
TLR-2 and MyD88-Dependent Activation of MAPK and STAT Proteins Regulates Proinflammatory Cytokine Response and Immunity to Experimental Trypanosoma congolense Infection.
Front Immunol
; 10: 2673, 2019.
Artículo
en Inglés
| MEDLINE | ID: mdl-31824484
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