Modification of ASC1 by UFM1 is crucial for ER&#945; transactivation and breast cancer development.

Yoo, Hee Min; Kang, Sung Hwan; Kim, Jae Yeon; Lee, Joo Eun; Seong, Min Woo; Lee, Seong Won; Ka, Seung Hyeun; Sou, Yu-Shin; Komatsu, Masaaki; Tanaka, Keiji; Lee, Soon Tae; Noh, Dong Young; Baek, Sung Hee; Jeon, Young Joo; Chung, Chin Ha

Modification of ASC1 by UFM1 is crucial for ERα transactivation and breast cancer development.

Yoo, Hee Min; Kang, Sung Hwan; Kim, Jae Yeon; Lee, Joo Eun; Seong, Min Woo; Lee, Seong Won; Ka, Seung Hyeun; Sou, Yu-Shin; Komatsu, Masaaki; Tanaka, Keiji; Lee, Soon Tae; Noh, Dong Young; Baek, Sung Hee; Jeon, Young Joo; Chung, Chin Ha.

Afiliación

Yoo HM; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Kang SH; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Kim JY; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Lee JE; Departments of Internal Medicine, Surgery, and Neurology, College of Medicine, Seoul National University, Seoul 110-744, Korea.
Seong MW; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Lee SW; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Ka SH; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Sou YS; Laboratory of Frontier Science, Tokyo Metropolitan Institute of Medical Science, Tokyo 113-8613, Japan.
Komatsu M; Laboratory of Frontier Science, Tokyo Metropolitan Institute of Medical Science, Tokyo 113-8613, Japan.
Tanaka K; Laboratory of Frontier Science, Tokyo Metropolitan Institute of Medical Science, Tokyo 113-8613, Japan.
Lee ST; Departments of Internal Medicine, Surgery, and Neurology, College of Medicine, Seoul National University, Seoul 110-744, Korea.
Noh DY; Departments of Internal Medicine, Surgery, and Neurology, College of Medicine, Seoul National University, Seoul 110-744, Korea.
Baek SH; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea.
Jeon YJ; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea. Electronic address: gydbs98@snu.ac.kr.
Chung CH; School of Biological Sciences, College of Natural Sciences, Seoul National University, Seoul 151-742, Korea. Electronic address: chchung@snu.ac.kr.

Mol Cell ; 56(2): 261-274, 2014 Oct 23.

Article en En | MEDLINE | ID: mdl-25219498

Asunto(s)

Sistema de Transporte de Aminoácidos y+/metabolismo; Neoplasias de la Mama/patología; Receptor alfa de Estrógeno/metabolismo; Proteínas/química; Sistema de Transporte de Aminoácidos y+/química; Sistema de Transporte de Aminoácidos y+/genética; Animales; Neoplasias de la Mama/metabolismo; Proteínas Portadoras/metabolismo; Línea Celular Tumoral; Cisteína Endopeptidasas/metabolismo; Proteína p300 Asociada a E1A/genética; Activación Enzimática/genética; Estradiol/genética; Estradiol/metabolismo; Antagonistas de Estrógenos/farmacología; Receptor alfa de Estrógeno/genética; Femenino; Células HEK293; Humanos; Células MCF-7; Ratones; Ratones Desnudos; Coactivador 1 de Receptor Nuclear/genética; Regiones Promotoras Genéticas/genética; Unión Proteica/genética; Proteínas/metabolismo; Tamoxifeno/farmacología; Activación Transcripcional; Ubiquitina/metabolismo; Enzimas Activadoras de Ubiquitina/genética; Ubiquitina-Proteína Ligasas/metabolismo

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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Proteínas / Sistema de Transporte de Aminoácidos y/ / Receptor alfa de Estrógeno Límite: Animals / Female / Humans Idioma: En Revista: Mol Cell Asunto de la revista: BIOLOGIA MOLECULAR Año: 2014 Tipo del documento: Article

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