Reciprocal modulation of Aß42 aggregation by copper and homocysteine.

Keskitalo, Salla; Farkas, Melinda; Hanenberg, Michael; Szodorai, Anita; Kulic, Luka; Semmler, Alexander; Weller, Michael; Nitsch, Roger M; Linnebank, Michael

Keskitalo, Salla; Farkas, Melinda; Hanenberg, Michael; Szodorai, Anita; Kulic, Luka; Semmler, Alexander; Weller, Michael; Nitsch, Roger M; Linnebank, Michael.

Afiliación

Keskitalo S; Department of Neurology, University Hospital Zurich Zurich, Switzerland.
Farkas M; Department of Neurology, University Hospital Zurich Zurich, Switzerland.
Hanenberg M; Division of Psychiatry Research, University of Zurich Schlieren, Switzerland.
Szodorai A; Division of Psychiatry Research, University of Zurich Schlieren, Switzerland.
Kulic L; Division of Psychiatry Research, University of Zurich Schlieren, Switzerland.
Semmler A; Department of Neurology, University Hospital Zurich Zurich, Switzerland.
Weller M; Department of Neurology, University Hospital Zurich Zurich, Switzerland.
Nitsch RM; Division of Psychiatry Research, University of Zurich Schlieren, Switzerland.
Linnebank M; Department of Neurology, University Hospital Zurich Zurich, Switzerland.

Front Aging Neurosci ; 6: 237, 2014.

Article en En | MEDLINE | ID: mdl-25249976

Palabras clave

Alzheimer's disease; Aß; copper; cytotoxicity; homocysteine; primary neurons

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Texto completo: 1 Banco de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Front Aging Neurosci Año: 2014 Tipo del documento: Article País de afiliación: Suiza

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