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Mutant WT1 is associated with DNA hypermethylation of PRC2 targets in AML and responds to EZH2 inhibition.
Sinha, Subarna; Thomas, Daniel; Yu, Linda; Gentles, Andrew J; Jung, Namyoung; Corces-Zimmerman, M Ryan; Chan, Steven M; Reinisch, Andreas; Feinberg, Andrew P; Dill, David L; Majeti, Ravindra.
Afiliación
  • Sinha S; Department of Computer Science, Stanford University, Stanford, CA;
  • Thomas D; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
  • Yu L; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
  • Gentles AJ; Department of Radiology, Stanford University, Stanford, CA; and.
  • Jung N; Center for Epigenetics, Institute for Basic Biomedical Science, Johns Hopkins School of Medicine, Baltimore, MD.
  • Corces-Zimmerman MR; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
  • Chan SM; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
  • Reinisch A; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
  • Feinberg AP; Center for Epigenetics, Institute for Basic Biomedical Science, Johns Hopkins School of Medicine, Baltimore, MD.
  • Dill DL; Department of Computer Science, Stanford University, Stanford, CA;
  • Majeti R; Division of Hematology, Department of Medicine, Cancer Institute, and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA;
Blood ; 125(2): 316-26, 2015 Jan 08.
Article en En | MEDLINE | ID: mdl-25398938
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Leucemia Mieloide Aguda / Regulación Leucémica de la Expresión Génica / Genes del Tumor de Wilms / Metilación de ADN / Perfilación de la Expresión Génica / Complejo Represivo Polycomb 2 Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: En Revista: Blood Año: 2015 Tipo del documento: Article
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Leucemia Mieloide Aguda / Regulación Leucémica de la Expresión Génica / Genes del Tumor de Wilms / Metilación de ADN / Perfilación de la Expresión Génica / Complejo Represivo Polycomb 2 Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: En Revista: Blood Año: 2015 Tipo del documento: Article