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Sustained Systemic Glucocerebrosidase Inhibition Induces Brain α-Synuclein Aggregation, Microglia and Complement C1q Activation in Mice.
Rocha, Emily M; Smith, Gaynor A; Park, Eric; Cao, Hongmei; Graham, Anne-Renee; Brown, Eilish; McLean, Jesse R; Hayes, Melissa A; Beagan, Jonathan; Izen, Sarah C; Perez-Torres, Eduardo; Hallett, Penelope J; Isacson, Ole.
Afiliación
  • Rocha EM; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Smith GA; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Park E; 2 Shire , Lexington, Massachusetts.
  • Cao H; 2 Shire , Lexington, Massachusetts.
  • Graham AR; 2 Shire , Lexington, Massachusetts.
  • Brown E; 2 Shire , Lexington, Massachusetts.
  • McLean JR; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Hayes MA; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Beagan J; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Izen SC; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Perez-Torres E; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Hallett PJ; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
  • Isacson O; 1 Neuroregeneration Research Institute, Harvard Medical School/McLean Hospital , Belmont, Massachusetts.
Antioxid Redox Signal ; 23(6): 550-64, 2015 Aug 20.
Article en En | MEDLINE | ID: mdl-26094487
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Complemento C1q / Microglía / Alfa-Sinucleína / Agregación Patológica de Proteínas / Glucosilceramidasa / Inositol Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Revista: Antioxid Redox Signal Asunto de la revista: METABOLISMO Año: 2015 Tipo del documento: Article
Texto completo
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Complemento C1q / Microglía / Alfa-Sinucleína / Agregación Patológica de Proteínas / Glucosilceramidasa / Inositol Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Revista: Antioxid Redox Signal Asunto de la revista: METABOLISMO Año: 2015 Tipo del documento: Article