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Histone Deacetylase SIRT1 Negatively Regulates the Differentiation of Interleukin-9-Producing CD4(+) T Cells.
Wang, Yu; Bi, Yujing; Chen, Xi; Li, Chunxiao; Li, Yan; Zhang, Zhengguo; Wang, Jian; Lu, Yun; Yu, Qing; Su, Huilin; Yang, Hui; Liu, Guangwei.
Afiliación
  • Wang Y; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Bi Y; State Key Laboratory of Pathogen and Biosecurity, Beijing Institute of Microbiology and Epidemiology, Beijing 100071, China.
  • Chen X; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Li C; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Li Y; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Zhang Z; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Wang J; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Lu Y; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Yu Q; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China.
  • Su H; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Yang H; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China.
  • Liu G; Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing 100875, China; Department of Immunology, School of Basic Medical Sciences, Fudan University, Shanghai 200032, China. Electroni
Immunity ; 44(6): 1337-49, 2016 06 21.
Article en En | MEDLINE | ID: mdl-27317260
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Linfocitos T Colaboradores-Inductores / Sirtuina 1 / Hipersensibilidad / Melanoma Límite: Animals / Humans Idioma: En Revista: Immunity Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article País de afiliación: China
Texto completo
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Linfocitos T Colaboradores-Inductores / Sirtuina 1 / Hipersensibilidad / Melanoma Límite: Animals / Humans Idioma: En Revista: Immunity Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article País de afiliación: China