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Autophagy Regulates Chromatin Ubiquitination in DNA Damage Response through Elimination of SQSTM1/p62.
Wang, Yanan; Zhang, Nan; Zhang, Luyao; Li, Ran; Fu, Wan; Ma, Ke; Li, Xue; Wang, Lina; Wang, Jiadong; Zhang, Hongquan; Gu, Wei; Zhu, Wei-Guo; Zhao, Ying.
Afiliación
  • Wang Y; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Zhang N; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Zhang L; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Li R; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Fu W; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Ma K; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Li X; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Wang L; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Wang J; Institute of Systems Biomedicine and Department of Radiation Medicine, School of Basic Medical Sciences, Peking University, Beijing 100191, China.
  • Zhang H; Department of Anatomy, Histology and Embryology, Peking University Health Science Center, Beijing 100191, China.
  • Gu W; Institute for Cancer Genetics and Department of Pathology and Cell Biology, College of Physicians and Surgeons, Columbia University, New York, NY 10032, USA.
  • Zhu WG; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
  • Zhao Y; Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Peking University Health Science Center, Beiji
Mol Cell ; 63(1): 34-48, 2016 07 07.
Article en En | MEDLINE | ID: mdl-27345151
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Autofagia / Cromatina / Neoplasias Colorrectales / Ensamble y Desensamble de Cromatina / Reparación del ADN / Roturas del ADN de Doble Cadena / Ubiquitinación / Proteína Sequestosoma-1 Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Mol Cell Asunto de la revista: BIOLOGIA MOLECULAR Año: 2016 Tipo del documento: Article
Texto completo
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Autofagia / Cromatina / Neoplasias Colorrectales / Ensamble y Desensamble de Cromatina / Reparación del ADN / Roturas del ADN de Doble Cadena / Ubiquitinación / Proteína Sequestosoma-1 Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Mol Cell Asunto de la revista: BIOLOGIA MOLECULAR Año: 2016 Tipo del documento: Article