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H2S Donor NaHS Changes the Production of Endogenous H2S and NO in D-Galactose-Induced Accelerated Ageing.
Wu, Wei; Hou, Cui-Lan; Mu, Xue-Pan; Sun, Chen; Zhu, Yi-Chun; Wang, Ming-Jie; Lv, Qian-Zhou.
Afiliación
  • Wu W; Department of Pharmacy, Zhongshan Hospital, Fudan University, Shanghai 200032, China.
  • Hou CL; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Shanghai Key Laboratory of Clinical Geriatric Medicine, Fudan University, Shanghai 200032, China.
  • Mu XP; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Shanghai Key Laboratory of Clinical Geriatric Medicine, Fudan University, Shanghai 200032, China.
  • Sun C; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Shanghai Key Laboratory of Clinical Geriatric Medicine, Fudan University, Shanghai 200032, China.
  • Zhu YC; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Shanghai Key Laboratory of Clinical Geriatric Medicine, Fudan University, Shanghai 200032, China.
  • Wang MJ; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Shanghai Key Laboratory of Clinical Geriatric Medicine, Fudan University, Shanghai 200032, China.
  • Lv QZ; Department of Pharmacy, Zhongshan Hospital, Fudan University, Shanghai 200032, China.
Oxid Med Cell Longev ; 2017: 5707830, 2017.
Article en En | MEDLINE | ID: mdl-28512525

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Sulfuros / Senescencia Celular / Galactosa / Sulfuro de Hidrógeno / Óxido Nítrico Tipo de estudio: Prognostic_studies Límite: Animals / Humans / Male Idioma: En Revista: Oxid Med Cell Longev Asunto de la revista: METABOLISMO Año: 2017 Tipo del documento: Article País de afiliación: China

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Sulfuros / Senescencia Celular / Galactosa / Sulfuro de Hidrógeno / Óxido Nítrico Tipo de estudio: Prognostic_studies Límite: Animals / Humans / Male Idioma: En Revista: Oxid Med Cell Longev Asunto de la revista: METABOLISMO Año: 2017 Tipo del documento: Article País de afiliación: China