Your browser doesn't support javascript.
loading
Genome-wide association studies suggest that APOL1-environment interactions more likely trigger kidney disease in African Americans with nondiabetic nephropathy than strong APOL1-second gene interactions.
Langefeld, Carl D; Comeau, Mary E; Ng, Maggie C Y; Guan, Meijian; Dimitrov, Latchezar; Mudgal, Poorva; Spainhour, Mitzie H; Julian, Bruce A; Edberg, Jeffrey C; Croker, Jennifer A; Divers, Jasmin; Hicks, Pamela J; Bowden, Donald W; Chan, Gary C; Ma, Lijun; Palmer, Nicholette D; Kimberly, Robert P; Freedman, Barry I.
Afiliación
  • Langefeld CD; Division of Public Health Sciences, Department of Biostatistical Sciences; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Public Health Genomics; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Comeau ME; Division of Public Health Sciences, Department of Biostatistical Sciences; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Public Health Genomics; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Ng MCY; Department of Biochemistry; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Guan M; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Dimitrov L; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Mudgal P; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Spainhour MH; Department of Internal Medicine, Section on Nephrology; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Julian BA; Department of Medicine, Division of Nephrology; University of Alabama at Birmingham School of Medicine, Birmingham, Alabama, USA.
  • Edberg JC; Department of Medicine, Division of Clinical Immunology and Rheumatology; University of Alabama at Birmingham School of Medicine, Birmingham, Alabama, USA.
  • Croker JA; Department of Medicine, Division of Clinical Immunology and Rheumatology; University of Alabama at Birmingham School of Medicine, Birmingham, Alabama, USA.
  • Divers J; Division of Public Health Sciences, Department of Biostatistical Sciences; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Public Health Genomics; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Hicks PJ; Department of Biochemistry; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Bowden DW; Department of Biochemistry; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Chan GC; Department of Internal Medicine, Section on Nephrology; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Ma L; Department of Internal Medicine, Section on Nephrology; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Palmer ND; Department of Biochemistry; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  • Kimberly RP; Department of Medicine, Division of Clinical Immunology and Rheumatology; University of Alabama at Birmingham School of Medicine, Birmingham, Alabama, USA.
  • Freedman BI; Center for Public Health Genomics; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Center for Diabetes Research; Wake Forest School of Medicine, Winston-Salem, North Carolina, USA; Department of Internal Medicine, Section on Nephrology; Wake Forest School of Medicine, Winston-Sal
Kidney Int ; 94(3): 599-607, 2018 09.
Article en En | MEDLINE | ID: mdl-29885931

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Negro o Afroamericano / Nefritis Lúpica / Interacción Gen-Ambiente / Apolipoproteína L1 / Fallo Renal Crónico Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies / Systematic_reviews Límite: Humans Idioma: En Revista: Kidney Int Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Negro o Afroamericano / Nefritis Lúpica / Interacción Gen-Ambiente / Apolipoproteína L1 / Fallo Renal Crónico Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies / Systematic_reviews Límite: Humans Idioma: En Revista: Kidney Int Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos