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PNPase knockout results in mtDNA loss and an altered metabolic gene expression program.
Shimada, Eriko; Ahsan, Fasih M; Nili, Mahta; Huang, Dian; Atamdede, Sean; TeSlaa, Tara; Case, Dana; Yu, Xiang; Gregory, Brian D; Perrin, Benjamin J; Koehler, Carla M; Teitell, Michael A.
Afiliación
  • Shimada E; Molecular Biology Institute Interdepartmental Program, University of California Los Angeles, Los Angeles, California, United States of America.
  • Ahsan FM; Department of Pathology and Laboratory Medicine, University of California Los Angeles, Los Angeles, California, United States of America.
  • Nili M; Department of Pathology and Laboratory Medicine, University of California Los Angeles, Los Angeles, California, United States of America.
  • Huang D; Department of Bioengineering, University of California Los Angeles, Los Angeles, California, United States of America.
  • Atamdede S; Department of Chemistry and Biochemistry, University of California Los Angeles, Los Angeles, California, United States of America.
  • TeSlaa T; Molecular Biology Institute Interdepartmental Program, University of California Los Angeles, Los Angeles, California, United States of America.
  • Case D; Molecular Biology Institute Interdepartmental Program, University of California Los Angeles, Los Angeles, California, United States of America.
  • Yu X; Department of Biology, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
  • Gregory BD; Department of Biology, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
  • Perrin BJ; Department of Biology, Indiana University-Purdue University Indianapolis, Indianapolis, Indiana, United States of America.
  • Koehler CM; Molecular Biology Institute Interdepartmental Program, University of California Los Angeles, Los Angeles, California, United States of America.
  • Teitell MA; Department of Chemistry and Biochemistry, University of California Los Angeles, Los Angeles, California, United States of America.
PLoS One ; 13(7): e0200925, 2018.
Article en En | MEDLINE | ID: mdl-30024931
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Polirribonucleótido Nucleotidiltransferasa / ADN Mitocondrial / Regulación de la Expresión Génica / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Pérdida Auditiva / Mitocondrias Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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PubMed Links
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Polirribonucleótido Nucleotidiltransferasa / ADN Mitocondrial / Regulación de la Expresión Génica / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Pérdida Auditiva / Mitocondrias Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos