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Acquired Expression of Mutant Mitofusin 2 Causes Progressive Neurodegeneration and Abnormal Behavior.
Ishikawa, Kaori; Yamamoto, Satoshi; Hattori, Satoko; Nishimura, Naoya; Tani, Haruna; Mito, Takayuki; Matsumoto, Hirokazu; Miyakawa, Tsuyoshi; Nakada, Kazuto.
Afiliación
  • Ishikawa K; Faculty of Life and Environmental Sciences, and k_ishikawa@biol.tsukuba.ac.jp knakada@biol.tsukuba.ac.jp.
  • Yamamoto S; Graduate School of Life and Environmental Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8572, Japan.
  • Hattori S; Integrated Technology Research Laboratories, Pharmaceutical Research Division, Takeda Pharmaceutical Company, Ltd, Fujisawa, Kanagawa, 251-8555, Japan, and.
  • Nishimura N; Division of Systems Medical Science, Institute for Comprehensive Medical Science, Fujita Health University, Toyoake, Aichi, 470-1192, Japan.
  • Tani H; Integrated Technology Research Laboratories, Pharmaceutical Research Division, Takeda Pharmaceutical Company, Ltd, Fujisawa, Kanagawa, 251-8555, Japan, and.
  • Mito T; Graduate School of Life and Environmental Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8572, Japan.
  • Matsumoto H; Faculty of Life and Environmental Sciences, and.
  • Miyakawa T; Integrated Technology Research Laboratories, Pharmaceutical Research Division, Takeda Pharmaceutical Company, Ltd, Fujisawa, Kanagawa, 251-8555, Japan, and.
  • Nakada K; Division of Systems Medical Science, Institute for Comprehensive Medical Science, Fujita Health University, Toyoake, Aichi, 470-1192, Japan.
J Neurosci ; 39(9): 1588-1604, 2019 02 27.
Article en En | MEDLINE | ID: mdl-30606759
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Encéfalo / Enfermedad de Charcot-Marie-Tooth / Mutación Missense / Proteínas Mitocondriales / GTP Fosfohidrolasas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: J Neurosci Año: 2019 Tipo del documento: Article
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Encéfalo / Enfermedad de Charcot-Marie-Tooth / Mutación Missense / Proteínas Mitocondriales / GTP Fosfohidrolasas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: J Neurosci Año: 2019 Tipo del documento: Article