A new insight into thymosin ß4, a promising therapeutic approach for neurodegenerative disorders.

Shomali, Navid; Baradaran, Behzad; Deljavanghodrati, Mina; Akbari, Morteza; Hemmatzadeh, Maryam; Mohammadi, Hamed; Jang, Yue; Xu, Huaxi; Sandoghchian Shotorbani, Siamak

Shomali, Navid; Baradaran, Behzad; Deljavanghodrati, Mina; Akbari, Morteza; Hemmatzadeh, Maryam; Mohammadi, Hamed; Jang, Yue; Xu, Huaxi; Sandoghchian Shotorbani, Siamak.

Afiliación

Shomali N; Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Baradaran B; Department of Immunology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Deljavanghodrati M; Student Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran.
Akbari M; Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Hemmatzadeh M; Department of Immunology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Mohammadi H; Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Jang Y; Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Xu H; Department of Immunology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Sandoghchian Shotorbani S; Non-Communicable Diseases Research Center, Alborz University of Medical Sciences, Karaj, Iran.

J Cell Physiol ; 235(4): 3270-3279, 2020 04.

Article en En | MEDLINE | ID: mdl-31612500

Asunto(s)

Inflamación/genética; Quinasas Asociadas a Receptores de Interleucina-1/genética; MicroARNs/genética; Enfermedades Neurodegenerativas/genética; Timosina/genética; Humanos; Inflamación/patología; Interleucina-1/genética; FN-kappa B/genética; Enfermedades Neurodegenerativas/patología; Transducción de Señal/genética; Receptores Toll-Like/genética

Palabras clave

CNS; TLR; Tß4; diseases; miR-146a; neurodegenerative disorder

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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Timosina / Enfermedades Neurodegenerativas / MicroARNs / Quinasas Asociadas a Receptores de Interleucina-1 / Inflamación Límite: Humans Idioma: En Revista: J Cell Physiol Año: 2020 Tipo del documento: Article País de afiliación: Irán

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