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NOS1AP polymorphisms reduce NOS1 activity and interact with prolonged repolarization in arrhythmogenesis.
Ronchi, Carlotta; Bernardi, Joyce; Mura, Manuela; Stefanello, Manuela; Badone, Beatrice; Rocchetti, Marcella; Crotti, Lia; Brink, Paul; Schwartz, Peter J; Gnecchi, Massimiliano; Zaza, Antonio.
Afiliación
  • Ronchi C; Department of Biotechnology and Biosciences, University of Milano-Bicocca, Piazza della Scienza 2, 2016 Milano, Italy.
  • Bernardi J; Department of Biotechnology and Biosciences, University of Milano-Bicocca, Piazza della Scienza 2, 2016 Milano, Italy.
  • Mura M; Department of Cardiothoracic and Vascular Sciences, Fondazione IRCCS Policlinico San Matteo - Laboratory of Experimental Cardiology for Cell and Molecular Therapies, Viale Camillo Golgi 19, 27100 Pavia, Italy.
  • Stefanello M; Department of Cardiothoracic and Vascular Sciences, Fondazione IRCCS Policlinico San Matteo - Laboratory of Experimental Cardiology for Cell and Molecular Therapies, Viale Camillo Golgi 19, 27100 Pavia, Italy.
  • Badone B; Department of Biotechnology and Biosciences, University of Milano-Bicocca, Piazza della Scienza 2, 2016 Milano, Italy.
  • Rocchetti M; Department of Biotechnology and Biosciences, University of Milano-Bicocca, Piazza della Scienza 2, 2016 Milano, Italy.
  • Crotti L; Center for Cardiac Arrhythmias of Genetic Origin, IRCCS Istituto Auxologico Italiano, Via Pier Lombardo 22, 20135 Milan, Italy.
  • Brink P; Department of Medicine and Surgery, University of Milano-Bicocca, Milano, Italy.
  • Schwartz PJ; Department of Cardiovascular, Neural and Metabolic Sciences, IRCCS Istituto Auxologico Italiano, San Luca Hospital, Milan, Italy.
  • Gnecchi M; Department of Medicine, University of Stellenbosch, Tygerberg, South Africa.
  • Zaza A; Center for Cardiac Arrhythmias of Genetic Origin, IRCCS Istituto Auxologico Italiano, Via Pier Lombardo 22, 20135 Milan, Italy.
Cardiovasc Res ; 117(2): 472-483, 2021 01 21.
Article en En | MEDLINE | ID: mdl-32061134
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Potenciales de Acción / Polimorfismo de Nucleótido Simple / Síndrome de Romano-Ward / Miocitos Cardíacos / Proteínas Adaptadoras Transductoras de Señales / Óxido Nítrico Sintasa de Tipo I / Canal de Potasio KCNQ1 / Células Madre Pluripotentes Inducidas / Frecuencia Cardíaca / Mutación Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Cardiovasc Res Año: 2021 Tipo del documento: Article País de afiliación: Italia
Texto completo
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XML
PubMed Links
Buscar en Google

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Potenciales de Acción / Polimorfismo de Nucleótido Simple / Síndrome de Romano-Ward / Miocitos Cardíacos / Proteínas Adaptadoras Transductoras de Señales / Óxido Nítrico Sintasa de Tipo I / Canal de Potasio KCNQ1 / Células Madre Pluripotentes Inducidas / Frecuencia Cardíaca / Mutación Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Cardiovasc Res Año: 2021 Tipo del documento: Article País de afiliación: Italia