RESUMO
UNLABELLED: CASE PRESENTATION AND SURGICAL INTERVENTION: a 3-year-old cat was presented with a recent history of dysphagia and intermittent regurgitation. Radiography revealed a bony oesophageal foreign body at the level of the thoracic inlet. Endoscopic retrieval was attempted but resulted in severe dyspnoea due to the development of pneumomediastinum, pneumothorax and subcutaneous emphysema secondary to perforation of the oesophageal wall. Immediate surgical exploration was carried out. Extensive necrosis of the oesophagus resulting from the presence of the foreign body led to a decision to perform an oesophageal resection and anastomosis. CLINICAL RELEVANCE: this is the first clinical report of a cat treated successfully by oesophagectomy following oesophageal perforation due to an obstructive foreign body. The authors suggest that prompt surgical intervention, the ability to convert to a surgical procedure under the same anaesthetic as a non-surgical retrieval, placement of a gastrostomy tube and the availability of advanced anaesthetic and critical care support are important factors to consider when managing feline patients with a perforating oesophageal foreign body.
Assuntos
Doenças do Gato/cirurgia , Perfuração Esofágica/veterinária , Corpos Estranhos/veterinária , Animais , Gatos , Perfuração Esofágica/etiologia , Perfuração Esofágica/cirurgia , Esofagectomia/veterinária , Feminino , Corpos Estranhos/complicações , Corpos Estranhos/cirurgia , Resultado do TratamentoRESUMO
BACKGROUND: In the veterinary literature, feline gastroduodenal ulcer disease has been described only in individual case reports or retrospective studies reporting small numbers of cats. Although its canine counterpart is commonly encountered, less is known about factors predisposing cats to gastroduodenal ulceration and potentially perforation. Similarly the outcome following treatment is not well reported in this species. REPORTED RISK FACTORS: In dogs, hepatic disease and administration of non-steroidal anti-inflammatory drugs (NSAIDs) have been reported as the two most common causes of gastroduodenal ulceration. In cats, only one clinical report so far has implicated an NSAID as a risk factor for spontaneous gastroduodenal perforation. FINDINGS AND PRACTICAL RELEVANCE: This report describes the historical and physical findings, as well as the treatment and outcome, in three cats with spontaneous gastric perforation that were receiving anti-inflammatory medication immediately prior to presentation. It highlights the importance of thorough patient evaluation in any cat presenting with non-specific clinical signs and a history of anti-inflammatory drug administration.
Assuntos
Anti-Inflamatórios não Esteroides/efeitos adversos , Doenças do Gato/cirurgia , Úlcera Péptica/veterinária , Gastropatias/veterinária , Animais , Anti-Inflamatórios não Esteroides/uso terapêutico , Doenças do Gato/induzido quimicamente , Doenças do Gato/diagnóstico , Gatos , Feminino , Masculino , Úlcera Péptica/induzido quimicamente , Úlcera Péptica/diagnóstico , Úlcera Péptica/cirurgia , Fatores de Risco , Gastropatias/induzido quimicamente , Gastropatias/diagnóstico , Gastropatias/cirurgia , Resultado do TratamentoRESUMO
Diaphragmatic defects in cats are common and most frequently occur as a result of trauma. Congenital diaphragmatic defects include peritoneopericardial hernias, hiatal hernias and, infrequently, true diaphragmatic, or pleuroperitoneal, hernias. Only three reports of feline pleuroperitoneal hernias could be found in the veterinary literature. All of these cats presented for evaluation of respiratory distress and two were managed successfully with surgery. This report describes the incidental diagnosis and successfully surgical treatment of a pleuroperitoneal hernia in a cat. It highlights the fact that pleuroperitoneal hernias may not always be symptomatic and that they may not be as rare as previously considered.