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Molecules ; 23(5)2018 May 18.
Artigo em Inglês | MEDLINE | ID: mdl-29783643

RESUMO

Lipopolysaccharide (LPS)-induced neuroinflammation triggers and accelerates the pathogenesis of Parkinson's disease (PD). Carthamus tinctorius L., a traditional Chinese medicine, has been widely used for the treatment of cerebrovascular disease. Hydroxysafflor Yellow A (HSYA) is an active component of C. tinctorius. The purpose of this study was to investigate whether HSYA could attenuate LPS-induced neurotoxicity and neuroinflammation in primary mesencephalic cultures. Cell viability was measured by MTT and LDH assays. The number of tyrosine hydroxylase (TH) positive neuron was observed by immunohistochemistry. NF-κB p65 and iNOS expressions were evaluated with western blotting method. Pro-inflammatory cytokines including IL-1ß and TNF-α were determined by ELISA kits. Nitric oxide (NO) content in the culture medium was assayed. The results showed that HSYA treatment significantly attenuated the LPS-induced dopaminergic neurons damage. HSYA partially inhibited the expressions of NF-κB p65 and iNOS. Furthermore, HSYA decreased the content of IL-1ß, TNF-α and NO in the supernatants. Taken together, these results suggest that HSYA exerts protective effects on LPS-induced neurotoxicity in dopaminergic neurons and the mechanisms may be associated with the inhibition of inflammatory response.


Assuntos
Chalcona/análogos & derivados , Inflamação/tratamento farmacológico , Lipopolissacarídeos/farmacologia , Neurônios/efeitos dos fármacos , Quinonas/farmacologia , Animais , Carthamus tinctorius/química , Sobrevivência Celular/efeitos dos fármacos , Células Cultivadas , Chalcona/química , Chalcona/farmacologia , Citocinas/metabolismo , Humanos , Mesencéfalo/citologia , Camundongos Endogâmicos C57BL , Neurônios/metabolismo , Óxidos de Nitrogênio , Cultura Primária de Células , Quinonas/química , Transdução de Sinais , Distribuição Tecidual , Fator de Transcrição RelA/metabolismo
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