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Silencing of the Drosophila ortholog of SOX5 leads to abnormal neuronal development and behavioral impairment.
Li, Airong; Hooli, Basavaraj; Mullin, Kristina; Tate, Rebecca E; Bubnys, Adele; Kirchner, Rory; Chapman, Brad; Hofmann, Oliver; Hide, Winston; Tanzi, Rudolph E.
Afiliación
  • Li A; Genetics and Aging Research Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases, Charlestown, MA 02129, USA.
  • Hooli B; Genetics and Aging Research Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases, Charlestown, MA 02129, USA.
  • Mullin K; Genetics and Aging Research Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases, Charlestown, MA 02129, USA.
  • Tate RE; Genetics and Aging Research Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases, Charlestown, MA 02129, USA.
  • Bubnys A; Genetics and Aging Research Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases, Charlestown, MA 02129, USA.
  • Kirchner R; Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
  • Chapman B; Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
  • Hofmann O; Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
  • Hide W; Center for Cancer Research, University of Melbourne, Melbourne 3000, Australia and.
  • Tanzi RE; Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
Hum Mol Genet ; 26(8): 1472-1482, 2017 04 15.
Article en En | MEDLINE | ID: mdl-28186563
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Texto completo: 1 Colección: 01-internacional Asunto principal: Discapacidades del Desarrollo / Proteínas de Drosophila / Factores de Transcripción SOXD / Enfermedad de Alzheimer / Esclerosis Amiotrófica Lateral Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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XML
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Texto completo: 1 Colección: 01-internacional Asunto principal: Discapacidades del Desarrollo / Proteínas de Drosophila / Factores de Transcripción SOXD / Enfermedad de Alzheimer / Esclerosis Amiotrófica Lateral Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos