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Loss of the Arp2/3 complex component ARPC1B causes platelet abnormalities and predisposes to inflammatory disease.
Kahr, Walter H A; Pluthero, Fred G; Elkadri, Abdul; Warner, Neil; Drobac, Marko; Chen, Chang Hua; Lo, Richard W; Li, Ling; Li, Ren; Li, Qi; Thoeni, Cornelia; Pan, Jie; Leung, Gabriella; Lara-Corrales, Irene; Murchie, Ryan; Cutz, Ernest; Laxer, Ronald M; Upton, Julia; Roifman, Chaim M; Yeung, Rae S M; Brumell, John H; Muise, Aleixo M.
Afiliación
  • Kahr WH; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Pluthero FG; Division of Haematology/Oncology, Department of Paediatrics, University of Toronto and The Hospital for Sick Children, Toronto, Ontario, Canada M5G 1X8.
  • Elkadri A; Department of Biochemistry, University of Toronto, Toronto, Ontario, Canada M5S 1A8.
  • Warner N; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Drobac M; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Chen CH; SickKids Inflammatory Bowel Disease Center and Division of Gastroenterology, Hepatology, and Nutrition, Department of Paediatrics, University of Toronto, Hospital for Sick Children, Toronto, Ontario, Canada M5G 1X8.
  • Lo RW; Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada M5S 1A8.
  • Li L; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Li R; SickKids Inflammatory Bowel Disease Center and Division of Gastroenterology, Hepatology, and Nutrition, Department of Paediatrics, University of Toronto, Hospital for Sick Children, Toronto, Ontario, Canada M5G 1X8.
  • Li Q; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Thoeni C; Department of Biochemistry, University of Toronto, Toronto, Ontario, Canada M5S 1A8.
  • Pan J; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Leung G; Department of Biochemistry, University of Toronto, Toronto, Ontario, Canada M5S 1A8.
  • Lara-Corrales I; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Murchie R; Department of Biochemistry, University of Toronto, Toronto, Ontario, Canada M5S 1A8.
  • Cutz E; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Laxer RM; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Upton J; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Roifman CM; SickKids Inflammatory Bowel Disease Center and Division of Gastroenterology, Hepatology, and Nutrition, Department of Paediatrics, University of Toronto, Hospital for Sick Children, Toronto, Ontario, Canada M5G 1X8.
  • Yeung RS; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
  • Brumell JH; SickKids Inflammatory Bowel Disease Center and Division of Gastroenterology, Hepatology, and Nutrition, Department of Paediatrics, University of Toronto, Hospital for Sick Children, Toronto, Ontario, Canada M5G 1X8.
  • Muise AM; Cell Biology Program, Research Institute, Hospital for Sick Children, Toronto, Ontario, Canada M5G 0A4.
Nat Commun ; 8: 14816, 2017 04 03.
Article en En | MEDLINE | ID: mdl-28368018
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Texto completo: 1 Colección: 01-internacional Asunto principal: Trastornos de las Plaquetas Sanguíneas / Plaquetas / Complejo 2-3 Proteico Relacionado con la Actina / Inflamación Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2017 Tipo del documento: Article
Texto completo
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Texto completo: 1 Colección: 01-internacional Asunto principal: Trastornos de las Plaquetas Sanguíneas / Plaquetas / Complejo 2-3 Proteico Relacionado con la Actina / Inflamación Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2017 Tipo del documento: Article