EPHA3 enhances macrophage autophagy and apoptosis by disrupting the mTOR signaling pathway in mice with endometriosis.

Xu, Hongmei; Gao, Yongmei; Shu, Yang; Wang, Yi; Shi, Qingyang

Xu, Hongmei; Gao, Yongmei; Shu, Yang; Wang, Yi; Shi, Qingyang.

Afiliación

Xu H; Department of Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Gao Y; Department of Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Shu Y; Department of Gynaecology and Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Wang Y; Department of Gynaecology and Obstetrics, The First Hospital of Jilin University, Changchun 130021, Jilin, China.
Shi Q; Centre for Reproductive Medicine, Centre for Prenatal Diagnosis, The First Hospital of Jilin University, Changchun 130021, Jilin, China shiqingyang1111@163.com.

Biosci Rep ; 39(7)2019 07 31.

Article en En | MEDLINE | ID: mdl-31262977

Asunto(s)

Apoptosis; Muerte Celular Autofágica; Endometriosis/metabolismo; Macrófagos/metabolismo; Receptor EphA3/metabolismo; Transducción de Señal; Serina-Treonina Quinasas TOR/metabolismo; Animales; Modelos Animales de Enfermedad; Endometriosis/genética; Endometriosis/patología; Femenino; Macrófagos/patología; Ratones; Ratones Endogámicos BALB C; Receptor EphA3/genética; Serina-Treonina Quinasas TOR/genética

Palabras clave

Apoptosis; Autophagy; EPHA3; Endometriosis; Macrophage; mTOR signaling pathway

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Texto completo: 1 Colección: 01-internacional Asunto principal: Transducción de Señal / Apoptosis / Receptor EphA3 / Endometriosis / Serina-Treonina Quinasas TOR / Muerte Celular Autofágica / Macrófagos Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Biosci Rep Año: 2019 Tipo del documento: Article País de afiliación: China

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