Vpr counteracts the restriction of LAPTM5 to promote HIV-1 infection in macrophages.

Zhao, Li; Wang, Shumei; Xu, Meng; He, Yang; Zhang, Xiaowei; Xiong, Ying; Sun, Hong; Ding, Haibo; Geng, Wenqing; Shang, Hong; Liang, Guoxin

Zhao, Li; Wang, Shumei; Xu, Meng; He, Yang; Zhang, Xiaowei; Xiong, Ying; Sun, Hong; Ding, Haibo; Geng, Wenqing; Shang, Hong; Liang, Guoxin.

Afiliación

Zhao L; Department of Laboratory Medicine, Key Laboratory of AIDS Immunology of Ministry of Health, The First Affiliated Hospital, China Medical University, Shenyang, China.
Wang S; National Clinical Research Center for Laboratory Medicine, The First Affiliated Hospital of China Medical University, Shenyang, China.
Xu M; Department of Laboratory Medicine, Key Laboratory of AIDS Immunology of Ministry of Health, The First Affiliated Hospital, China Medical University, Shenyang, China.
He Y; National Clinical Research Center for Laboratory Medicine, The First Affiliated Hospital of China Medical University, Shenyang, China.
Zhang X; Research Institute for Cancer Therapy, The First Affiliated Hospital, China Medical University, Shenyang, China.
Xiong Y; Research Institute for Cancer Therapy, The First Affiliated Hospital, China Medical University, Shenyang, China.
Sun H; Department of Laboratory Medicine, Key Laboratory of AIDS Immunology of Ministry of Health, The First Affiliated Hospital, China Medical University, Shenyang, China.
Ding H; National Clinical Research Center for Laboratory Medicine, The First Affiliated Hospital of China Medical University, Shenyang, China.
Geng W; Department of Laboratory Medicine, Key Laboratory of AIDS Immunology of Ministry of Health, The First Affiliated Hospital, China Medical University, Shenyang, China.
Shang H; National Clinical Research Center for Laboratory Medicine, The First Affiliated Hospital of China Medical University, Shenyang, China.
Liang G; Department of Laboratory Medicine, Key Laboratory of AIDS Immunology of Ministry of Health, The First Affiliated Hospital, China Medical University, Shenyang, China.

Nat Commun ; 12(1): 3691, 2021 06 17.

Article en En | MEDLINE | ID: mdl-34140527

Asunto(s)

Infecciones por VIH/metabolismo; VIH-1/metabolismo; Interacciones Microbiota-Huesped; Macrófagos/metabolismo; Macrófagos/virología; Proteínas de la Membrana/metabolismo; Productos del Gen vpr del Virus de la Inmunodeficiencia Humana/metabolismo; Linfocitos T CD4-Positivos/metabolismo; Linfocitos T CD4-Positivos/virología; Silenciador del Gen; Infecciones por VIH/genética; Infecciones por VIH/virología; VIH-1/genética; VIH-1/patogenicidad; VIH-2/metabolismo; VIH-2/patogenicidad; Interacciones Microbiota-Huesped/genética; Humanos; Lisosomas/metabolismo; Proteínas de la Membrana/genética; Proteínas Serina-Treonina Quinasas/metabolismo; Estabilidad Proteica; Virus de la Inmunodeficiencia de los Simios/metabolismo; Virus de la Inmunodeficiencia de los Simios/patogenicidad; Ubiquitina-Proteína Ligasas/metabolismo; Regulación hacia Arriba; Virión/metabolismo

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Texto completo: 1 Colección: 01-internacional Asunto principal: Infecciones por VIH / VIH-1 / Productos del Gen vpr del Virus de la Inmunodeficiencia Humana / Interacciones Microbiota-Huesped / Macrófagos / Proteínas de la Membrana Límite: Humans Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2021 Tipo del documento: Article País de afiliación: China

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