microRNA-149-5p mediates the PM<sub>2.5</sub>-induced inflammatory response by targeting TAB2 via MAPK and NF-&#954;B signaling pathways in vivo and in vitro.

Li, Qiuyue; Li, Siling; Xu, Chunjie; Zhao, Jing; Hou, Lin; Jiang, Fuyang; Zhu, Zhonghui; Wang, Yan; Tian, Lin

microRNA-149-5p mediates the PM_2.5-induced inflammatory response by targeting TAB2 via MAPK and NF-κB signaling pathways in vivo and in vitro.

Li, Qiuyue; Li, Siling; Xu, Chunjie; Zhao, Jing; Hou, Lin; Jiang, Fuyang; Zhu, Zhonghui; Wang, Yan; Tian, Lin.

Afiliación

Li Q; Department of Occupational and Environmental Health, School of Public Health, Capital Medical University, No. 10, Xitoutiao Youanmen Street, Beijing, 100069, China.
Li S; Beijing Key Laboratory of Environmental Toxicology, Capital Medical University, Beijing, 100069, China.
Xu C; Department of Occupational and Environmental Health, School of Public Health, Capital Medical University, No. 10, Xitoutiao Youanmen Street, Beijing, 100069, China.
Zhao J; Beijing Key Laboratory of Environmental Toxicology, Capital Medical University, Beijing, 100069, China.
Hou L; Department of Occupational and Environmental Health, School of Public Health, Capital Medical University, No. 10, Xitoutiao Youanmen Street, Beijing, 100069, China.
Jiang F; Beijing Key Laboratory of Environmental Toxicology, Capital Medical University, Beijing, 100069, China.
Zhu Z; Department of Occupational and Environmental Health, School of Public Health, Capital Medical University, No. 10, Xitoutiao Youanmen Street, Beijing, 100069, China.
Wang Y; Beijing Key Laboratory of Environmental Toxicology, Capital Medical University, Beijing, 100069, China.
Tian L; Department of Occupational and Environmental Health, School of Public Health, Capital Medical University, No. 10, Xitoutiao Youanmen Street, Beijing, 100069, China.

Cell Biol Toxicol ; 39(3): 703-717, 2023 06.

Article en En | MEDLINE | ID: mdl-34331613

Asunto(s)

MicroARNs; Enfermedad Pulmonar Obstructiva Crónica; Humanos; Ratones; Animales; FN-kappa B/metabolismo; Proteínas Quinasas Activadas por Mitógenos/metabolismo; MicroARNs/genética; MicroARNs/metabolismo; Transducción de Señal; Material Particulado/toxicidad; Inflamación/genética; Inflamación/patología; Enfermedad Pulmonar Obstructiva Crónica/genética; Enfermedad Pulmonar Obstructiva Crónica/metabolismo; Proteínas Adaptadoras Transductoras de Señales/genética; Proteínas Adaptadoras Transductoras de Señales/metabolismo

Palabras clave

COPD; Inflammatory response; PM2.5; TAB2; miR-149-5p

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Texto completo: 1 Colección: 01-internacional Asunto principal: Enfermedad Pulmonar Obstructiva Crónica / MicroARNs Límite: Animals / Humans Idioma: En Revista: Cell Biol Toxicol Asunto de la revista: TOXICOLOGIA Año: 2023 Tipo del documento: Article País de afiliación: China

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