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HSC70 mediated autophagic degradation of oxidized PRL2 is responsible for osteoclastogenesis and inflammatory bone destruction.
Li, Qi; Yue, Tao; Du, Xinyue; Tang, Zaiming; Cui, Jinjie; Wang, Weifeng; Xia, Wenjie; Ren, Baiyang; Kan, Shuo; Li, Chang; Wu, Chenyun; Niu, Xiaoyin; Li, Bin; Lin, Kaili; Luo, Jian; Chen, Guangjie; Wang, Zhaojun.
Afiliación
  • Li Q; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Yue T; Department of Rheumatology, Guanghua Hospital Affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, 200052, China.
  • Du X; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Tang Z; Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Department of Pathophysiology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Cui J; Department of Oral and Cranio-Maxillofacial Surgery, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200011, China.
  • Wang W; Department of Rheumatology, Guanghua Hospital Affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, 200052, China.
  • Xia W; Department of Rheumatology, Guanghua Hospital Affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, 200052, China.
  • Ren B; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Kan S; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Li C; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Wu C; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Niu X; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Li B; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China.
  • Lin K; Department of Oral and Cranio-Maxillofacial Surgery, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200011, China.
  • Luo J; Yangzhi Rehabilitation Hospital (Sunshine Rehabilitation Center), Tongji University School of Medicine, Shanghai, 201619, China.
  • Chen G; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China. guangjie_chen@163.com.
  • Wang Z; Department of Immunology and Microbiology, Shanghai Institute of Immunology, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China. zjwang@sjtu.edu.cn.
Cell Death Differ ; 30(3): 647-659, 2023 03.
Article en En | MEDLINE | ID: mdl-36182990
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Texto completo: 1 Colección: 01-internacional Asunto principal: Osteoporosis / Resorción Ósea Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Cell Death Differ Año: 2023 Tipo del documento: Article País de afiliación: China
Texto completo
Imprimir
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PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Asunto principal: Osteoporosis / Resorción Ósea Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Cell Death Differ Año: 2023 Tipo del documento: Article País de afiliación: China