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BMC Vet Res ; 17(1): 350, 2021 Nov 16.
Artigo em Inglês | MEDLINE | ID: mdl-34784920

RESUMO

BACKGROUND: Gentamicin (GM) is a low-cost, low-resistance antibiotic commonly used to treat gram-negative bacterial diseases. Cisplatin (Csp) is a platinum-derived anti-neoplastic agent. This experiment aimed to identify the early signs of gentamicin and cisplatin-induced nephrotoxicity in rats. Thirty Wistar rats were divided into three groups of 10: a control group, which received no treatment; a gentamicin group administered by a dose of (100 mg/kg, IP) for 7 consecutive days, and a cisplatin group was administered intraperitoneal in a dose of (1.5 mg/kg body weight) repeated twice a week for 3 weeks. RESULTS: Both experimental groups exhibited increased levels of creatinine, urea, and uric acid, with the cisplatin-treated group showing higher levels than the gentamicin group. Experimental groups also exhibited significantly increased Malondialdehyde (MDA), reduced glutathione (GSH), and glutathione peroxidase (GSH-Px) with more pronounced effects in the cisplatin-treated group. Further, both experimental groups exhibited significant up-regulation of Tumor Necrosis Factor α (TNF-α), caspase-3, and Bax and down regulation of Bcl-2. CONCLUSION: These findings confirm the use of necrotic, apoptotic genes as early biomarkers in the detection of tubular kidney damage. Further, cisplatin was shown to have a greater nephrotoxic effect than gentamicin; therefore, its use should be constrained accordingly when co-administered with gentamicin.


Assuntos
Cisplatino/toxicidade , Gentamicinas/toxicidade , Nefropatias/induzido quimicamente , Animais , Antibacterianos/toxicidade , Antineoplásicos/toxicidade , Apoptose/genética , Biomarcadores , Caspase 3/genética , Genes bcl-2/genética , Nefropatias/patologia , Masculino , Necrose/genética , Ratos Wistar , Fator de Necrose Tumoral alfa/genética , Proteína X Associada a bcl-2/genética
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