Detalhe da pesquisa
1.
Apobec2 deficiency causes mitochondrial defects and mitophagy in skeletal muscle.
FASEB J
; 32(3): 1428-1439, 2018 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-29127187
2.
CTNNBL1 facilitates the association of CWC15 with CDC5L and is required to maintain the abundance of the Prp19 spliceosomal complex.
Nucleic Acids Res
; 43(14): 7058-69, 2015 Aug 18.
Artigo
em Inglês
| MEDLINE | ID: mdl-26130721
3.
AID and RPA: PKA makes the connection local.
Nat Immunol
; 10(4): 367-9, 2009 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-19295635
4.
Interaction between antibody-diversification enzyme AID and spliceosome-associated factor CTNNBL1.
Mol Cell
; 31(4): 474-484, 2008 Aug 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-18722174
5.
The cytoplasmic AID complex.
Semin Immunol
; 24(4): 273-80, 2012 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-22698843
6.
Somatic hypermutation at A.T pairs: polymerase error versus dUTP incorporation.
Nat Rev Immunol
; 5(2): 171-8, 2005 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-15688043
7.
Uracil excision by endogenous SMUG1 glycosylase promotes efficient Ig class switching and impacts on A:T substitutions during somatic mutation.
Eur J Immunol
; 44(7): 1925-35, 2014 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-24771041
8.
Germline ablation of SMUG1 DNA glycosylase causes loss of 5-hydroxymethyluracil- and UNG-backup uracil-excision activities and increases cancer predisposition of Ung-/-Msh2-/- mice.
Nucleic Acids Res
; 40(13): 6016-25, 2012 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-22447450
9.
Cytoplasmic activation-induced cytidine deaminase (AID) exists in stoichiometric complex with translation elongation factor 1α (eEF1A).
Proc Natl Acad Sci U S A
; 108(45): 18366-71, 2011 Nov 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-22042842
10.
Somatic hypermutation: activation-induced deaminase for C/G followed by polymerase eta for A/T.
J Exp Med
; 204(1): 7-10, 2007 Jan 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-17190841
11.
Mutagenesis by AID: Being in the Right Place at the Right Time.
PLoS Genet
; 11(9): e1005489, 2015 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-26355597
12.
Erratum: Enhancement of Adeno-Associated Virus-Mediated Gene Therapy Using Hydroxychloroquine in Murine and Human Tissues.
Mol Ther Methods Clin Dev
; 28: 300, 2023 Mar 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-36843676
13.
CTNNBL1 is a novel nuclear localization sequence-binding protein that recognizes RNA-splicing factors CDC5L and Prp31.
J Biol Chem
; 286(19): 17091-102, 2011 May 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-21385873
14.
The in vivo pattern of AID targeting to immunoglobulin switch regions deduced from mutation spectra in msh2-/- ung-/- mice.
J Exp Med
; 203(9): 2085-94, 2006 Sep 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-16894013
15.
The dependence of Ig class-switching on the nuclear export sequence of AID likely reflects interaction with factors additional to Crm1 exportin.
Eur J Immunol
; 41(2): 485-90, 2011 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-21268017
16.
The stability of AID and its function in class-switching are critically sensitive to the identity of its nuclear-export sequence.
Proc Natl Acad Sci U S A
; 106(16): 6736-41, 2009 Apr 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-19351893
17.
Deficiency in APOBEC2 leads to a shift in muscle fiber type, diminished body mass, and myopathy.
J Biol Chem
; 285(10): 7111-8, 2010 Mar 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-20022958
18.
In memoriam: Jefferson Foote.
MAbs
; 13(1): 1870059, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-33435811
19.
Erratum: The uracil-DNA glycosylase UNG protects the fitness of normal and cancer B cells expressing AID.
NAR Cancer
; 3(1): zcaa045, 2021 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-34316697
20.
The AKV murine leukemia virus is restricted and hypermutated by mouse APOBEC3.
J Virol
; 83(22): 11550-9, 2009 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-19726503