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1.
De novo loss-of-function KCNMA1 variants are associated with a new multiple malformation syndrome and a broad spectrum of developmental and neurological phenotypes.
Hum Mol Genet
; 28(17): 2937-2951, 2019 09 01.
Article
in English
| MEDLINE | ID: mdl-31152168
2.
Ubiquitination-activating enzymes UBE1 and UBA6 regulate ubiquitination and expression of cardiac sodium channel Nav1.5.
Biochem J
; 477(9): 1683-1700, 2020 05 15.
Article
in English
| MEDLINE | ID: mdl-32315024
3.
UBC9 regulates cardiac sodium channel Nav1.5 ubiquitination, degradation and sodium current density.
J Mol Cell Cardiol
; 129: 79-91, 2019 04.
Article
in English
| MEDLINE | ID: mdl-30772377
4.
Mechanistic insights into the interaction of the MOG1 protein with the cardiac sodium channel Nav1.5 clarify the molecular basis of Brugada syndrome.
J Biol Chem
; 293(47): 18207-18217, 2018 11 23.
Article
in English
| MEDLINE | ID: mdl-30282806
5.
14-3-3ε/YWHAE regulates the transcriptional expression of cardiac sodium channel NaV1.5.
Heart Rhythm
; 2024 May 13.
Article
in English
| MEDLINE | ID: mdl-38750908
6.
Immune Cell Infiltration Analysis Based on Bioinformatics Reveals Novel Biomarkers of Coronary Artery Disease.
J Inflamm Res
; 16: 3169-3184, 2023.
Article
in English
| MEDLINE | ID: mdl-37525634
7.
Comprehensive Analysis of circRNA-miRNA-mRNA Regulatory Network and Novel Potential Biomarkers in Acute Myocardial Infarction.
Front Cardiovasc Med
; 9: 850991, 2022.
Article
in English
| MEDLINE | ID: mdl-35872921
8.
Expression pattern and diagnostic value of ferroptosis-related genes in acute myocardial infarction.
Front Cardiovasc Med
; 9: 993592, 2022.
Article
in English
| MEDLINE | ID: mdl-36407421
9.
Small GTPases SAR1A and SAR1B regulate the trafficking of the cardiac sodium channel Nav1.5.
Biochim Biophys Acta Mol Basis Dis
; 1864(11): 3672-3684, 2018 11.
Article
in English
| MEDLINE | ID: mdl-30251687
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