Pesquisa | Portal de Pesquisa da BVS Enfermagem

1.

Discovery of Hippo signaling as a regulator of CSPG4 expression and as a therapeutic target for Clostridioides difficile disease.

Larabee, Jason L; Doyle, D Annie; Ahmed, Ummey Khalecha Bintha; Shadid, Tyler M; Sharp, Rachel R; Jones, Kenneth L; Kim, Young Mi; Li, Shibo; Ballard, Jimmy D.

PLoS Pathog ; 19(3): e1011272, 2023 03.

Artigo em Inglês | MEDLINE | ID: mdl-36972308

2.

Recommendations for future university pandemic responses: What the first COVID-19 shutdown taught us.

Coyne, Carolyn; Ballard, Jimmy D; Blader, Ira J.

PLoS Biol ; 18(8): e3000889, 2020 08.

Artigo em Inglês | MEDLINE | ID: mdl-32853196

3.

Response Regulator CD1688 Is a Negative Modulator of Sporulation in Clostridioides difficile.

Kempher, Megan L; Morris, Savannah C; Shadid, Tyler M; Menon, Smita K; Ballard, Jimmy D; West, Ann H.

J Bacteriol ; 204(8): e0013022, 2022 08 16.

Artigo em Inglês | MEDLINE | ID: mdl-35852332

4.

Identification of ClpP Dual Isoform Disruption as an Antisporulation Strategy for Clostridioides difficile.

Bishop, Catherine E; Shadid, Tyler M; Lavey, Nathan P; Kempher, Megan L; Ballard, Jimmy D; Duerfeldt, Adam S.

J Bacteriol ; 204(2): e0041121, 2022 02 15.

Artigo em Inglês | MEDLINE | ID: mdl-34807726

5.

Clostridioides difficile Toxin B Activates Group 3 Innate Lymphocytes.

Pope, Rosemary L; Chitrakar, Alisha; Sah, Prakash; Shadid, Tyler; Ballard, Jimmy D; Zenewicz, Lauren A.

Infect Immun ; 90(4): e0007322, 2022 04 21.

Artigo em Inglês | MEDLINE | ID: mdl-35377172

6.

The Murine Neonatal Fc Receptor Is Required for Transport of Immunization-Induced C. difficile-Specific IgG to the Gut and Protection against Disease but Does Not Affect Disease Susceptibility.

Amadou Amani, Souwelimatou; Lang, Gillian A; Ballard, Jimmy D; Lang, Mark L.

Infect Immun ; 89(10): e0027421, 2021 09 16.

Artigo em Inglês | MEDLINE | ID: mdl-34097471

7.

α-Galactosylceramide-Reactive NKT Cells Increase IgG1 Class Switch against a Clostridioides difficile Polysaccharide Antigen and Enhance Immunity against a Live Pathogen Challenge.

Lang, Gillian A; Shrestha, Binu; Amadou Amani, Souwelimatou; Shadid, Tyler M; Ballard, Jimmy D; Lang, Mark L.

Infect Immun ; 89(11): e0043821, 2021 10 15.

Artigo em Inglês | MEDLINE | ID: mdl-34424751

8.

Clostridioides difficile Infection Induces an Inferior IgG Response to That Induced by Immunization and Is Associated with a Lack of T Follicular Helper Cell and Memory B Cell Expansion.

Amadou Amani, Souwelimatou; Shadid, Tyler; Ballard, Jimmy D; Lang, Mark L.

Infect Immun ; 88(3)2020 02 20.

Artigo em Inglês | MEDLINE | ID: mdl-31871095

9.

Cell-penetrating peptides derived from Clostridium difficile TcdB2 and a related large clostridial toxin.

Larabee, Jason L; Hauck, Garrett D; Ballard, Jimmy D.

J Biol Chem ; 293(5): 1810-1819, 2018 02 02.

Artigo em Inglês | MEDLINE | ID: mdl-29247010

10.

Deletion of a 19-Amino-Acid Region in Clostridioides difficile TcdB2 Results in Spontaneous Autoprocessing and Reduced Cell Binding and Provides a Nontoxic Immunogen for Vaccination.

Bland, Sarah J; Larabee, Jason L; Shadid, Tyler M; Lang, Mark L; Ballard, Jimmy D.

Infect Immun ; 87(8)2019 08.

Artigo em Inglês | MEDLINE | ID: mdl-31138612

11.

Pathogens boosted by food additive.

Ballard, Jimmy D.

Nature ; 553(7688): 285-286, 2018 Jan.

Artigo em Inglês | MEDLINE | ID: mdl-32094593

12.

Pathogens boosted by food additive.

Ballard, Jimmy D.

Nature ; 553(7688): 285-286, 2018 01 18.

Artigo em Inglês | MEDLINE | ID: mdl-29345660

13.

Exposure of neutralizing epitopes in the carboxyl-terminal domain of TcdB is altered by a proximal hypervariable region.

Larabee, Jason L; Krumholz, Aleze; Hunt, Jonathan J; Lanis, Jordi M; Ballard, Jimmy D.

J Biol Chem ; 290(11): 6975-85, 2015 Mar 13.

Artigo em Inglês | MEDLINE | ID: mdl-25614625

14.

Memory B Cells Encode Neutralizing Antibody Specific for Toxin B from the Clostridium difficile Strains VPI 10463 and NAP1/BI/027 but with Superior Neutralization of VPI 10463 Toxin B.

Devera, T Scott; Lang, Gillian A; Lanis, Jordi M; Rampuria, Pragya; Gilmore, Casey L; James, Judith A; Ballard, Jimmy D; Lang, Mark L.

Infect Immun ; 84(1): 194-204, 2016 01.

Artigo em Inglês | MEDLINE | ID: mdl-26502913

15.

Clostridium difficile 027/BI/NAP1 encodes a hypertoxic and antigenically variable form of TcdB.

Lanis, Jordi M; Heinlen, Latisha D; James, Judith A; Ballard, Jimmy D.

PLoS Pathog ; 9(8): e1003523, 2013.

Artigo em Inglês | MEDLINE | ID: mdl-23935501

16.

Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE).

Larabee, Jason L; Shakir, Salika M; Barua, Soumitra; Ballard, Jimmy D.

J Biol Chem ; 288(30): 21526-36, 2013 Jul 26.

Artigo em Inglês | MEDLINE | ID: mdl-23775085

17.

CSPG4-dependent cytotoxicity for C. difficile TcdB is influenced by extracellular calcium and chondroitin sulfate.

Doyle, D Annie; DeAngelis, Paul L; Ballard, Jimmy D.

mSphere ; 9(4): e0009424, 2024 Apr 23.

Artigo em Inglês | MEDLINE | ID: mdl-38470254

18.

Clostridioides difficile toxin B subverts germinal center and antibody recall responses by stimulating a drug-treatable CXCR4-dependent mechanism.

Norman, Kaylee M; Lang, Gillian A; Shadid, Tyler M; Honold, Sydney T; Reel, Jessica M; Cox, Maureen A; Ballard, Jimmy D; Lang, Mark L.

Cell Rep ; 43(5): 114245, 2024 May 28.

Artigo em Inglês | MEDLINE | ID: mdl-38761377

19.

Toxin inhibition of antimicrobial factors induced by Bacillus anthracis peptidoglycan in human blood.

Barua, Soumitra; Iyer, Janaki K; Larabee, Jason L; Raisley, Brent; Hughes, Molly A; Coggeshall, K Mark; Ballard, Jimmy D.

Infect Immun ; 81(10): 3693-702, 2013 Oct.

Artigo em Inglês | MEDLINE | ID: mdl-23876807

20.

TcdB from hypervirulent Clostridium difficile exhibits increased efficiency of autoprocessing.

Lanis, Jordi M; Hightower, Logan D; Shen, Aimee; Ballard, Jimmy D.

Mol Microbiol ; 84(1): 66-76, 2012 Apr.

Artigo em Inglês | MEDLINE | ID: mdl-22372854

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Discovery of Hippo signaling as a regulator of CSPG4 expression and as a therapeutic target for Clostridioides difficile disease.

Recommendations for future university pandemic responses: What the first COVID-19 shutdown taught us.

Response Regulator CD1688 Is a Negative Modulator of Sporulation in Clostridioides difficile.

Identification of ClpP Dual Isoform Disruption as an Antisporulation Strategy for Clostridioides difficile.

Clostridioides difficile Toxin B Activates Group 3 Innate Lymphocytes.

The Murine Neonatal Fc Receptor Is Required for Transport of Immunization-Induced C. difficile-Specific IgG to the Gut and Protection against Disease but Does Not Affect Disease Susceptibility.

α-Galactosylceramide-Reactive NKT Cells Increase IgG1 Class Switch against a Clostridioides difficile Polysaccharide Antigen and Enhance Immunity against a Live Pathogen Challenge.

Clostridioides difficile Infection Induces an Inferior IgG Response to That Induced by Immunization and Is Associated with a Lack of T Follicular Helper Cell and Memory B Cell Expansion.

Cell-penetrating peptides derived from Clostridium difficile TcdB2 and a related large clostridial toxin.

Deletion of a 19-Amino-Acid Region in Clostridioides difficile TcdB2 Results in Spontaneous Autoprocessing and Reduced Cell Binding and Provides a Nontoxic Immunogen for Vaccination.

Pathogens boosted by food additive.

Pathogens boosted by food additive.

Exposure of neutralizing epitopes in the carboxyl-terminal domain of TcdB is altered by a proximal hypervariable region.

Memory B Cells Encode Neutralizing Antibody Specific for Toxin B from the Clostridium difficile Strains VPI 10463 and NAP1/BI/027 but with Superior Neutralization of VPI 10463 Toxin B.

Clostridium difficile 027/BI/NAP1 encodes a hypertoxic and antigenically variable form of TcdB.

Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE).

CSPG4-dependent cytotoxicity for C. difficile TcdB is influenced by extracellular calcium and chondroitin sulfate.

Clostridioides difficile toxin B subverts germinal center and antibody recall responses by stimulating a drug-treatable CXCR4-dependent mechanism.

Toxin inhibition of antimicrobial factors induced by Bacillus anthracis peptidoglycan in human blood.

TcdB from hypervirulent Clostridium difficile exhibits increased efficiency of autoprocessing.