Detalhe da pesquisa
1.
Incomplete-penetrant hypertrophic cardiomyopathy MYH7 G256E mutation causes hypercontractility and elevated mitochondrial respiration.
Proc Natl Acad Sci U S A
; 121(19): e2318413121, 2024 May 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-38683993
2.
Edge Strand Dissociation and Conformational Changes in Transthyretin under Amyloidogenic Conditions.
Biophys J
; 119(10): 1995-2009, 2020 11 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-33091379
3.
Myosin's powerstroke transitions define atomic scale movement of cardiac thin filament tropomyosin.
J Gen Physiol
; 156(5)2024 May 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-38607351
4.
Hypertrophic cardiomyopathy mutations Y115H and E497D disrupt the folded-back state of human beta-cardiac myosin allosterically.
bioRxiv
; 2024 Mar 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-38464145
5.
Multi-scale models reveal hypertrophic cardiomyopathy MYH7 G256E mutation drives hypercontractility and elevated mitochondrial respiration.
bioRxiv
; 2023 Jun 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-37333118
6.
Post-translational modification patterns on ß-myosin heavy chain are altered in ischemic and nonischemic human hearts.
Elife
; 112022 05 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-35502901
7.
Conserved patterns and interactions in the unfolding transition state across SH3 domain structural homologues.
Protein Sci
; 30(2): 391-407, 2021 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-33190305