Detalhe da pesquisa
1.
Exogenous ubiquitin attenuates hypoxia/reoxygenation-induced cardiac myocyte apoptosis via the involvement of CXCR4 and modulation of mitochondrial homeostasis.
Biochem Cell Biol
; 98(4): 492-501, 2020 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-31967865
2.
Exogenous ubiquitin reduces inflammatory response and preserves myocardial function 3 days post-ischemia-reperfusion injury.
Am J Physiol Heart Circ Physiol
; 316(3): H617-H628, 2019 03 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30681370
3.
Ataxia telangiectasia-mutated kinase deficiency exacerbates left ventricular dysfunction and remodeling late after myocardial infarction.
Am J Physiol Heart Circ Physiol
; 311(2): H445-52, 2016 08 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27288435
4.
Osteopontin stimulates apoptosis in adult cardiac myocytes via the involvement of CD44 receptors, mitochondrial death pathway, and endoplasmic reticulum stress.
Am J Physiol Heart Circ Physiol
; 306(8): H1182-91, 2014 Apr 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24531809
5.
Extracellular ubiquitin increases expression of angiogenic molecules and stimulates angiogenesis in cardiac microvascular endothelial cells.
Microcirculation
; 21(4): 324-32, 2014 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-24308702
6.
Exogenous ubiquitin modulates chronic ß-adrenergic receptor-stimulated myocardial remodeling: role in Akt activity and matrix metalloproteinase expression.
Am J Physiol Heart Circ Physiol
; 303(12): H1459-68, 2012 Dec 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-23042947
7.
Extracellular ubiquitin modulates cardiac fibroblast phenotype and function via its interaction with CXCR4.
Life Sci
; 211: 8-16, 2018 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-30195032
8.
Deficiency of ataxia telangiectasia mutated kinase delays inflammatory response in the heart following myocardial infarction.
J Am Heart Assoc
; 3(6): e001286, 2014 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-25520329
9.
Deficiency of ataxia telangiectasia mutated kinase modulates cardiac remodeling following myocardial infarction: involvement in fibrosis and apoptosis.
PLoS One
; 8(12): e83513, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-24358288