Detalhe da pesquisa
1.
Blocking PD-L1-PD-1 improves senescence surveillance and ageing phenotypes.
Nature
; 611(7935): 358-364, 2022 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-36323784
2.
Application of targeted nanopore sequencing for the screening and determination of structural variants in patients with Lynch syndrome.
J Hum Genet
; 66(11): 1053-1060, 2021 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-33958709
3.
Development of an MSI-positive colon tumor with aberrant DNA methylation in a PPAP patient.
J Hum Genet
; 64(8): 729-740, 2019 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-31089268
4.
Pseudomyxoma peritonei of a mature ovarian teratoma caused by mismatch repair deficiency in a patient with Lynch syndrome: a case report.
BMC Med Genet
; 17(1): 94, 2016 Dec 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-27938333
5.
Detection of APC mosaicism by next-generation sequencing in an FAP patient.
J Hum Genet
; 60(5): 227-31, 2015 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-25716913
6.
M2 macrophage-derived TGF-ß induces age-associated loss of adipogenesis through progenitor cell senescence.
Mol Metab
; 84: 101943, 2024 Apr 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-38657734
7.
KMT2C expression and DNA homologous recombination repair factors in lung cancers with a high-grade fetal adenocarcinoma component.
Transl Lung Cancer Res
; 12(8): 1738-1751, 2023 Aug 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-37691868
8.
Comprehensive molecular analysis of genomic profiles and PD-L1 expression in lung adenocarcinoma with a high-grade fetal adenocarcinoma component.
Transl Lung Cancer Res
; 10(3): 1292-1304, 2021 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-33889510
9.
Senolysis by glutaminolysis inhibition ameliorates various age-associated disorders.
Science
; 371(6526): 265-270, 2021 01 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-33446552
10.
Generation of a p16 Reporter Mouse and Its Use to Characterize and Target p16high Cells In Vivo.
Cell Metab
; 32(5): 814-828.e6, 2020 11 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-32949498
11.
Reduced expression of APC-1B but not APC-1A by the deletion of promoter 1B is responsible for familial adenomatous polyposis.
Sci Rep
; 6: 26011, 2016 05 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-27217144
12.
Attenuated familial adenomatous polyposis with desmoids caused by an APC mutation.
Hum Genome Var
; 2: 15011, 2015.
Artigo
em Inglês
| MEDLINE | ID: mdl-27081525