Detalhe da pesquisa
1.
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
PLoS Biol
; 15(8): e2001992, 2017 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-28771465
2.
Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.
Nucleic Acids Res
; 45(5): 2368-2383, 2017 03 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-27903901
3.
Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.
PLoS Pathog
; 11(7): e1005031, 2015 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-26153983
4.
Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.
PLoS Pathog
; 9(2): e1003187, 2013 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-23436997
5.
BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV.
Nucleic Acids Res
; 40(15): 7233-46, 2012 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-22584624
6.
Two nonconsensus sites in the Epstein-Barr virus oncoprotein EBNA3A cooperate to bind the co-repressor carboxyl-terminal-binding protein (CtBP).
J Biol Chem
; 277(49): 47197-204, 2002 Dec 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-12372828