Detalhe da pesquisa
1.
Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis.
Circ Res
; 134(5): 505-525, 2024 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-38422177
2.
Ketone Body Exposure of Cardiomyocytes Impairs Insulin Sensitivity and Contractile Function through Vacuolar-Type H+-ATPase Disassembly-Rescue by Specific Amino Acid Supplementation.
Int J Mol Sci
; 23(21)2022 Oct 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-36361698
3.
Metabolic Interventions to Prevent Hypertrophy-Induced Alterations in Contractile Properties In Vitro.
Int J Mol Sci
; 22(7)2021 Mar 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-33807195
4.
Putative Role of Protein Palmitoylation in Cardiac Lipid-Induced Insulin Resistance.
Int J Mol Sci
; 21(24)2020 Dec 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-33322406
5.
Specific amino acid supplementation rescues the heart from lipid overload-induced insulin resistance and contractile dysfunction by targeting the endosomal mTOR-v-ATPase axis.
Mol Metab
; 53: 101293, 2021 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-34265467