Detalhe da pesquisa
1.
Methyl-Metabolite Depletion Elicits Adaptive Responses to Support Heterochromatin Stability and Epigenetic Persistence.
Mol Cell
; 78(2): 210-223.e8, 2020 04 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-32208170
2.
Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency.
J Virol
; 91(5)2017 03 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-28003489
3.
5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.
Proc Natl Acad Sci U S A
; 112(52): E7257-65, 2015 Dec 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-26663912
4.
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells.
PLoS Pathog
; 11(10): e1005195, 2015 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-26431332
5.
Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replication.
J Virol
; 87(2): 935-50, 2013 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-23135711
6.
DOT1L interaction partner AF10 controls patterning of H3K79 methylation and RNA polymerase II to maintain cell identity.
Stem Cell Reports
; 18(12): 2451-2463, 2023 12 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-37995701
7.
DOT1L is a barrier to histone acetylation during reprogramming to pluripotency.
Sci Adv
; 9(46): eadf3980, 2023 11 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-37976354
8.
Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency.
J Virol
; 85(17): 8940-53, 2011 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-21697476
9.
Connecting the DOTs on Cell Identity.
Front Cell Dev Biol
; 10: 906713, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-35733849
10.
DOT1L inhibition enhances pluripotency beyond acquisition of epithelial identity and without immediate suppression of the somatic transcriptome.
Stem Cell Reports
; 17(2): 384-396, 2022 02 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-34995500