Detalhe da pesquisa
1.
The central adaptor molecule TRIF influences L. sigmodontis worm development.
Parasitol Res
; 118(2): 539-549, 2019 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-30643971
2.
Absence of IL-17A in Litomosoides sigmodontis-infected mice influences worm development and drives elevated filarial-specific IFN-γ.
Parasitol Res
; 117(8): 2665-2675, 2018 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-29931394
3.
Adoptive Transfer of Immune Cells Into RAG2IL-2Rγ-Deficient Mice During Litomosoides sigmodontis Infection: A Novel Approach to Investigate Filarial-Specific Immune Responses.
Front Immunol
; 12: 777860, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-34868049
4.
Filarial Lymphedema Patients Are Characterized by Exhausted CD4+ T Cells.
Front Cell Infect Microbiol
; 11: 767306, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-35071034
5.
Distinct Immune Profiles of Exhausted Effector and Memory CD8+ T Cells in Individuals With Filarial Lymphedema.
Front Cell Infect Microbiol
; 11: 680832, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-34485170
6.
Multivariable Regression Analysis in Schistosoma mansoni-Infected Individuals in the Sudan Reveals Unique Immunoepidemiological Profiles in Uninfected, egg+ and Non-egg+ Infected Individuals.
PLoS Negl Trop Dis
; 10(5): e0004629, 2016 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-27152725
7.
Development of patent Litomosoides sigmodontis infections in semi-susceptible C57BL/6 mice in the absence of adaptive immune responses.
Parasit Vectors
; 8: 396, 2015 Jul 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-26209319