Detalhe da pesquisa
1.
SOCS3 Ablation in Leptin Receptor-Expressing Cells Causes Autonomic and Cardiac Dysfunctions in Middle-Aged Mice despite Improving Energy and Glucose Metabolism.
Int J Mol Sci
; 23(12)2022 Jun 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-35742928
2.
Downregulation of the vascular renin-angiotensin system by aerobic training - focus on the balance between vasoconstrictor and vasodilator axes - .
Circ J
; 79(6): 1372-80, 2015.
Artigo
em Inglês
| MEDLINE | ID: mdl-25808225
3.
Hypothalamic Kisspeptin Neurons as a Target for Whole-Cell Patch-Clamp Recordings.
J Vis Exp
; (193)2023 03 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-37010276
4.
Acute effects of somatomammotropin hormones on neuronal components of the hypothalamic-pituitary-gonadal axis.
Brain Res
; 1714: 210-217, 2019 07 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30851245
5.
Exercise training increases GAD65 expression, restores the depressed GABAA receptor function within the PVN and reduces sympathetic modulation in hypertension.
Physiol Rep
; 7(13): e14107, 2019 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-31264387
6.
Central growth hormone signaling is not required for the timing of puberty.
J Endocrinol
; 2019 08 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-31470413
7.
Suppression of Prolactin Secretion Partially Explains the Antidiabetic Effect of Bromocriptine in ob/ob Mice.
Endocrinology
; 160(1): 193-204, 2019 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30462197
8.
Maternal metabolic adaptations are necessary for normal offspring growth and brain development.
Physiol Rep
; 6(5)2018 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-29536670
9.
STAT5 signaling in kisspeptin cells regulates the timing of puberty.
Mol Cell Endocrinol
; 448: 55-65, 2017 06 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28344041
10.
Fatness rather than leptin sensitivity determines the timing of puberty in female mice.
Mol Cell Endocrinol
; 423: 11-21, 2016 Mar 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-26762764
11.
Obesity impairs lactation performance in mice by inducing prolactin resistance.
Sci Rep
; 6: 22421, 2016 Mar 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26926925
12.
Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice.
Endocrinology
; 157(10): 3901-3914, 2016 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-27471877
13.
Reviewing the Effects of L-Leucine Supplementation in the Regulation of Food Intake, Energy Balance, and Glucose Homeostasis.
Nutrients
; 7(5): 3914-37, 2015 May 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-26007339
14.
SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes.
Mol Metab
; 4(3): 237-45, 2015 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-25737950
15.
L-leucine supplementation worsens the adiposity of already obese rats by promoting a hypothalamic pattern of gene expression that favors fat accumulation.
Nutrients
; 6(4): 1364-73, 2014 Apr 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-24699194
16.
Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice.
Endocrinology
; 155(11): 4226-36, 2014 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-25144922
17.
Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus.
PLoS One
; 8(12): e84094, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-24349566