Growing up with Bilateral parieto-occipital injury: over ten years of clinical observation and neuropsychological follow-up.

This paper presents a follow-up of a child with Balint's syndrome over more than a decade. The patient experienced traumatic brain injury before age 12, resulting in bilateral occipito-parietal infarctions and a clinical presentation of Balint's syndrome. Neuropsychological assessments at three time points showed average verbal abilities alongside persistent difficulties in visual orientation, mirrored in the patient's daily life. Her outstanding compensatory abilities in the face of these impairments are discussed with respect to the recruitment of the ventral visual stream and the role of top-down processing. This profile may help to determine interventions for younger patients with similar lesions.

Palinopsia Accompanied with Migraine Attack After Having Occipital Brain Abscess.

Palinopsia is defined as persistence of recurrent visual images after the stimulus has been removed. A 55-year-old male patient with long-standing migraine history developed brain abscess in the right occipital lobe. The patient developed episodes of palinopsia accompanied with migraine attack after healing of occipital lesion. The phenomenon cannot be sufficiently explained with single risk factor, although occipital lesion and migraine are both known to cause palinopsia. We speculated that migrainous brain is not always symptomatic but may drive cortex sensitive for second insult, such as occipital brain lesion. The predisposition may contribute to appearance of palinopsia.

Unusual visual loss in a patient with exudative macular degeneration.

PURPOSE: To describe a case of marked vision loss in a patient with neovascular age-related macular degeneration after choroidal neovascular membrane (CNV) improvement and stabilization. OBSERVATIONS: An 82-year-old male presented with 20/800 vision having dropped from 20/50 three months prior. He had been undergoing active treatment for exudative macular degeneration over the past seven years, the CNV had stabilized. An extensive ophthalmic workup was performed revealing no CNV progression and no ophthalmic cause was identified for visual loss. An MRI of the brain was obtained, which showed a metastatic brain lesion in the occipital lobe, and subsequent workup determined it originated from an adenosquamous carcinoma of the lung. CONCLUSIONS: When there is unexplained visual loss in an otherwise stable patient with macular degeneration, suspicion for non-retinal related causes of visual loss could alter the morbidity and mortality for patients with systemic diseases.