TrkA gene ablation in basal forebrain results in dysfunction of the cholinergic circuitry.

Sanchez-Ortiz, Efrain; Yui, Daishi; Song, Dongli; Li, Yun; Rubenstein, John L; Reichardt, Louis F; Parada, Luis F

Sanchez-Ortiz, Efrain; Yui, Daishi; Song, Dongli; Li, Yun; Rubenstein, John L; Reichardt, Louis F; Parada, Luis F.

Affiliation

Sanchez-Ortiz E; Department of Developmental Biology, University of Texas Southwestern Medical School, Dallas, TX 75390-9133, USA.

J Neurosci ; 32(12): 4065-79, 2012 Mar 21.

Article in En | MEDLINE | ID: mdl-22442072

Subject(s)

Cholinergic Neurons/physiology; Prosencephalon; Receptor, trkA/deficiency; Amino Acids/metabolism; Analysis of Variance; Animals; Animals, Newborn; Cell Count; Cell Size; Choline O-Acetyltransferase/metabolism; Cholinergic Neurons/ultrastructure; Conditioning, Psychological/physiology; Dopamine and cAMP-Regulated Phosphoprotein 32/metabolism; Embryo, Mammalian; Fear/physiology; Homeodomain Proteins/genetics; Homeodomain Proteins/metabolism; Maze Learning/physiology; Mice; Mice, Inbred C57BL; Mice, Knockout; Prosencephalon/cytology; Prosencephalon/embryology; Prosencephalon/growth & development; Proteins/genetics; RNA, Untranslated; Receptor, Nerve Growth Factor/metabolism; Receptor, trkA/genetics; Recognition, Psychology/physiology; Signal Transduction/genetics; Signal Transduction/physiology; Silver Staining

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Full text: 1 Database: MEDLINE Main subject: Prosencephalon / Receptor, trkA / Cholinergic Neurons Limits: Animals Language: En Journal: J Neurosci Year: 2012 Type: Article Affiliation country: United States

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