Cutting Edge: Brucella abortus exploits a cellular prion protein on intestinal M cells as an invasive receptor.

Nakato, Gaku; Hase, Koji; Suzuki, Michio; Kimura, Masanobu; Ato, Manabu; Hanazato, Misaho; Tobiume, Minoru; Horiuchi, Motohiro; Atarashi, Ryuichiro; Nishida, Noriyuki; Watarai, Masahisa; Imaoka, Koichi; Ohno, Hiroshi

Nakato, Gaku; Hase, Koji; Suzuki, Michio; Kimura, Masanobu; Ato, Manabu; Hanazato, Misaho; Tobiume, Minoru; Horiuchi, Motohiro; Atarashi, Ryuichiro; Nishida, Noriyuki; Watarai, Masahisa; Imaoka, Koichi; Ohno, Hiroshi.

Affiliation

Nakato G; Laboratory for Epithelial Immunobiology, Research Center for Allergy and Immunology, RIKEN, Kanagawa 230-0045, Japan;

J Immunol ; 189(4): 1540-4, 2012 Aug 15.

Article in En | MEDLINE | ID: mdl-22772447

Subject(s)

Brucella abortus/metabolism; Brucellosis/metabolism; Intestinal Mucosa/metabolism; PrPC Proteins/metabolism; Animals; Brucella abortus/pathogenicity; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Peyer's Patches/metabolism; Reverse Transcriptase Polymerase Chain Reaction

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google

Full text: 1 Database: MEDLINE Main subject: Brucella abortus / Brucellosis / PrPC Proteins / Intestinal Mucosa Limits: Animals Language: En Journal: J Immunol Year: 2012 Type: Article

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google