Loss of caveolin-1 expression in knock-in mouse model of Huntington's disease suppresses pathophysiology in vivo.

Trushina, Eugenia; Canaria, Christie A; Lee, Do-Yup; McMurray, Cynthia T

Trushina, Eugenia; Canaria, Christie A; Lee, Do-Yup; McMurray, Cynthia T.

Affiliation

Trushina E; Department of Neurology and.

Hum Mol Genet ; 23(1): 129-44, 2014 Jan 01.

Article in En | MEDLINE | ID: mdl-24021477

Subject(s)

Caveolin 1/genetics; Caveolin 1/metabolism; Cholesterol/metabolism; Huntington Disease/genetics; Huntington Disease/pathology; Nerve Tissue Proteins/genetics; Nerve Tissue Proteins/metabolism; Animals; Cell Membrane/metabolism; Disease Models, Animal; Endoplasmic Reticulum/metabolism; Fluorescence Resonance Energy Transfer; Gene Knock-In Techniques; HEK293 Cells; Humans; Huntingtin Protein; Mice; Mice, Inbred C57BL; Mice, Transgenic; Neurons/metabolism; Neurons/pathology; Nuclear Proteins/genetics; Nuclear Proteins/metabolism; Phenotype

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Full text: 1 Database: MEDLINE Main subject: Cholesterol / Huntington Disease / Caveolin 1 / Nerve Tissue Proteins Limits: Animals / Humans Language: En Journal: Hum Mol Genet Journal subject: BIOLOGIA MOLECULAR / GENETICA MEDICA Year: 2014 Type: Article

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