Periodic mechanical stress stimulates the FAK mitogenic signal in rat chondrocytes through ERK1/2 activity.

Liang, Wenwei; Ren, Kewei; Liu, Feng; Cui, Weiding; Wang, Qing; Chen, Zhefeng; Fan, Weimin

Liang, Wenwei; Ren, Kewei; Liu, Feng; Cui, Weiding; Wang, Qing; Chen, Zhefeng; Fan, Weimin.

Affiliation

Liang W; Department of Orthopedics, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China Co-first author.

Cell Physiol Biochem ; 32(4): 915-30, 2013.

Article in En | MEDLINE | ID: mdl-24217647

Subject(s)

Chondrocytes/cytology; Chondrocytes/metabolism; Focal Adhesion Protein-Tyrosine Kinases/metabolism; Mitogen-Activated Protein Kinase 1/metabolism; Mitogen-Activated Protein Kinase 3/metabolism; Stress, Mechanical; Animals; Blotting, Western; Cell Proliferation; Cells, Cultured; Female; Focal Adhesion Protein-Tyrosine Kinases/genetics; Male; Mitogen-Activated Protein Kinase 1/genetics; Mitogen-Activated Protein Kinase 3/genetics; Rats; Rats, Sprague-Dawley; Real-Time Polymerase Chain Reaction; Signal Transduction/genetics; Signal Transduction/physiology

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Full text: 1 Database: MEDLINE Main subject: Stress, Mechanical / Chondrocytes / Mitogen-Activated Protein Kinase 1 / Mitogen-Activated Protein Kinase 3 / Focal Adhesion Protein-Tyrosine Kinases Limits: Animals Language: En Journal: Cell Physiol Biochem Journal subject: BIOQUIMICA / FARMACOLOGIA Year: 2013 Type: Article

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